Objectives: Various cytokines induced by inhalation of coal dust may mediate inflammation
and lead to tissue damage or fibrosis, such as coal workers' pneumoconiosis (CWP) …

Various cytokines activated by the inhalation of coal dust may mediate inflammation and
lead to tissue damage. Objective of this study was to examine the relationships between …

It is well known that various cytokines and growth factors secreted from macrophages/
monocytes play the key role in the pathogenesis of pneumoconiosis. These can act as …

The aim of this study was to investigate whether systemic tumor necrosis factor alpha (TNF-
α), soluble TNF-α receptors (p55, p75), interleukin 6 (IL-6), and soluble IL-6 receptor could …

It has been shown that coal dust exposure stimulates inflammatory response leading to
increased release of cytokines from monocytes such as TNF-alpha and IL1. These released …

Inhaled inorganic dusts such as coal can cause inflammation and fibrosis in the lung called
pneumoconiosis. Chronic inflammatory process in the lung is associated with various …

Coal workers' pneumoconiosis (CWP) is an occupational pulmonary disease that occurs by
chronic inhalation of coal dust. CWP is divided into two stages depending on the extent of …

Objective To evaluate the alterations of biomarkers in the development and progression of
coal workers' pneumoconiosis (CWP). Methods The type and number of cells, and the levels …

Objective: To evaluate the component of coal responsible for coal workers' pneumoconiosis
(CWP). Methods: A literature search of PubMED was conducted to address studies that have …

Background and objective Coal mine dust has a complex and heterogeneous chemical
composition. It has been suggested that coal particle chemistry plays a critical role in …