Background The level of environmental hypobaric hypoxia that affects climbers at the
summit of Mount Everest (8848 m [29,029 ft]) is close to the limit of tolerance by humans. We …

Background High altitude pulmonary oedema (HAPE) that is severe enough to require
urgent medical care is infrequent. We hypothesised that subclinical HAPE is far more …

In five men with a history of susceptibility to high-altitude pulmonary edema (HAPE),
hemodynamics and pulmonary gas exchange were measured at sea level, and again 24 …

Breathing against positive expiratory pressure has been used to improve gas exchange in
many forms of pulmonary edema, and forced expiration against resistance during exercise …

Abstract Karinen, Heikki, Juha E. Peltonen, Mika Kähönen, and Heikki O. Tikkanen.
Prediction of acute mountain sickness by monitoring arterial saturation during ascent. High …

Reduced tolerance to high altitude may be associated with a low ventilatory and an
increased pulmonary vascular response to hypoxia. We therefore, examined whether …

Individuals susceptible to high-altitude pulmonary oedema (HAPE) are characterised by an
abnormal increase of pulmonary artery systolic pressure (PASP) in hypoxia and during …

Background Acute mountain sickness (AMS) is a usually self-limiting syndrome
encompassing headache, nausea and dizziness. AMS is seen in those that go from low to …

High-altitude pulmonary edema (HAPE), a severe form of altitude illness that can occur in
young healthy individuals, is a noncardiogenic form of edema that is associated with high …

A very high ventilatory response to hypoxia is believed necessary to reach extreme altitude
without oxygen. Alternatively, the excessive ventilation could be counterproductive by …