Closing the gap on drug-induced liver injury

M Maurel, J Rosenbaum - Hepatology, 2012 - journals.lww.com
Drug-induced liver injury (DILI) limits the development and application of many therapeutic
compounds and presents major challenges to the pharmaceutical industry and clinical …

Gap junction inhibition prevents drug-induced liver toxicity and fulminant hepatic failure

SJ Patel, JM Milwid, KR King, S Bohr… - Nature …, 2012 - nature.com
Drug-induced liver injury (DILI) limits the development and application of many therapeutic
compounds and presents major challenges to the pharmaceutical industry and clinical …

[HTML][HTML] Involvement of connexin43 in acetaminophen-induced liver injury

M Maes, MR McGill, TC da Silva, C Abels… - … et Biophysica Acta (BBA …, 2016 - Elsevier
Background and aims Being goalkeepers of liver homeostasis, gap junctions are also
involved in hepatotoxicity. However, their role in this process is ambiguous, as gap junctions …

Gap junction dysfunction reduces acetaminophen hepatotoxicity with impact on apoptotic signaling and connexin 43 protein induction in rat

A Naiki-Ito, M Asamoto, T Naiki, K Ogawa… - Toxicologic …, 2010 - journals.sagepub.com
Acetaminophen (APAP) is a widely used antipyretic and analgesic agent. However,
overdosing and sometimes even a recommended dose can lead to serious and conceivably …

Connexin hemichannel inhibition reduces acetaminophen-induced liver injury in mice

M Maes, SC Yanguas, J Willebrords, JL Weemhoff… - Toxicology letters, 2017 - Elsevier
Historically, connexin hemichannels have been considered as structural precursors of gap
junctions. However, accumulating evidence points to independent roles for connexin …

Connexin32: a mediator of acetaminophen-induced liver injury?

M Maes, MR McGill, TC Da Silva… - Toxicology …, 2016 - Taylor & Francis
Connexin32 is the building block of hepatocellular gap junctions, which control direct
intercellular communication and thereby act as goalkeepers of liver homeostasis. This study …

The gap junction inhibitor 2-aminoethoxy-diphenyl-borate protects against acetaminophen hepatotoxicity by inhibiting cytochrome P450 enzymes and c-jun N-terminal …

K Du, CD Williams, MR McGill, Y Xie, A Farhood… - Toxicology and applied …, 2013 - Elsevier
Acetaminophen (APAP) hepatotoxicity is the leading cause of acute liver failure in the US.
Although many aspects of the mechanism are known, recent publications suggest that gap …

Hepatic Gap Junction Inhibition Limits Liver Injury and Inflammation in Non-alcoholic Steatohepatitis (NASH): 50

SJ Patel, J Luther, KR King, S Bohr, J Scichilone… - Hepatology, 2013 - journals.lww.com
Suraj J. Patel1, 3, Jay Luther2, Kevin R. King1, Stefan Bohr1, 3, John Scichilone1, 3, John
Garber2, Lee F. Peng2, Raymond T. Chung2, Martin L. Yarmush1, 3; 1Surgery …

Connexin 32 deficiency protects the liver against ischemia/reperfusion injury

S Wu, W Yao, C Chen, H Chen, F Huang, Y Liu… - European Journal of …, 2020 - Elsevier
Hepatic ischemia/reperfusion (I/R) injury is a common complication in the clinical setting.
Our previous study has shown that connexin 32 (Cx32) plays a major role in renal I/R injury; …

[HTML][HTML] Role of connexin 32 in acetaminophen toxicity in a knockout mice model

I Igarashi, T Maejima, K Kai, S Arakawa… - Experimental and …, 2014 - Elsevier
Gap junctional intercellular communication (GJIC), by which glutathione (GSH) and
inorganic ions are transmitted to neighboring cells, is recognized as being largely involved …