Focal brain damage after aneurysmal subarachnoid haemorrhage predominantly results
from intracerebral haemorrhage, and early and delayed cerebral ischaemia. The …

Objective To investigate whether spreading depolarization (SD)-related variables at 2
different time windows (days 1–4 and 5–8) after aneurysmal subarachnoid hemorrhage …

Progressive ischaemic damage in animals is associated with spreading mass
depolarizations of neurons and astrocytes, detected as spreading negative slow voltage …

Importance Advances in treatment of traumatic brain injury are hindered by the inability to
monitor pathological mechanisms in individual patients for targeted neuroprotective …

It has been hypothesized that vasospasm is the prime mechanism of delayed cerebral
ischemia (DCI) after aneurysmal subarachnoid hemorrhage (aSAH). Recently, it was found …

Abstract See Ghoshal and Claassen (doi: 10.1093/brain/awx226) for a scientific commentary
on this article. Early cortical infarcts are common in poor-grade patients after aneurysmal …

Objective Delayed ischemic neurological deficit (DIND) contributes to poor outcome in
subarachnoid hemorrhage (SAH) patients. Because there is continuing uncertainty as to …

Spreading depolarization of cells in cerebral grey matter is characterized by massive ion
translocation, neuronal swelling and large changes in direct current-coupled voltage …

Background and Purpose—We studied the dynamics of extracellular brain tissue
concentrations of glucose, lactate, pyruvate, and glutamate during the occurrence of …

Cortical spreading depolarizations occur spontaneously after ischaemic, haemorrhagic and
traumatic brain injury. Their effects vary spatially and temporally as graded phenomena …