Background Myasthenia gravis (MG) and the experimental autoimmune MG (EAMG) animal
model are characterized by T-cell-induced and B-cell-dominated autoimmune diseases that …

Myasthenia gravis (MG) is a T cell-dependent, B-cell mediated autoimmune disease caused
by antibodies against the nicotinic acetylcholine receptor or other components of the post …

Myasthenia gravis (MG) is a T cell-driven, B cell-mediated and autoantibody-dependent
autoimmune disorder against neuromuscular junctions (NMJ). Accumulated evidence has …

The weakness in myasthenia gravis (MG) is mediated by T helper cell (Th)‐dependent
autoantibodies against neuromuscular epitopes. So far, analyzing Th phenotypes or antigen …

Myasthenia gravis (MG) is an autoimmune neuromuscular disorder characterized by
impaired neuromuscular transmission, which causes fluctuating fatigable muscle weakness …

Myasthenia gravis (MG) is an autoimmune disease mediated by autoantibodies
predominantly against the acetylcholine receptor (AChR). Specific T cell subsets are …

Background In myasthenia gravis (MG) patients, the dysfunction of CD4+ CD25+ regulatory
T cells (CD4+ CD25+ Tregs) may be one of the important pathogenesis of MG. Currently, the …

Myasthenia gravis (MG) is a chronic humoral immunity–mediated autoimmune disorder of
the neuromuscular junction characterized by muscle weakness. Follicular helper T (Tfh) …

T regulatory cells (Tregs) are crucial for the development of self-tolerance and are the major
focus in many studies interpreting the pathogenesis of myasthenia gravis (MG), an …

Introduction: The changes in the number and function of regulatory T cells (Tregs) are
thought to play important roles in the pathogenesis of generalized myasthenia gravis (gMG) …