G-protein activated inwardly rectifying K+ channels (GIRKs) of the heterotetrameric
GIRK1/GIRK4 composition mediate IK+ ACh in atrium and are regulated by cAMP …

The cardiac G protein-gated K+ channel, I KACh, is directly activated by G protein βγ
subunits (G βγ). I KACh is composed of two inward rectifier K+ channel subunits, GIRK1 and …

G-protein-regulated inwardly rectifying K+(GIRK) channels play critical inhibitory roles
throughout the nervous system, heart, and pancreas. They are believed to be …

G protein-gated inwardly rectifying K+ (GIRK) channels, which are important regulators of
membrane excitability both in heart and brain, appear to function as heteromultimers. GIRK1 …

The inwardly rectifying K+ channels of the GIRK (Kir3) family, members of the superfamily of
inwardly rectifying K+ channels (Kir), are important physiological tools to regulate excitability …

G-protein-coupled inwardly rectifying K+(GIRK; Kir3. x) channels are the primary effectors of
numerous G-protein-coupled receptors. GIRK channels decrease cellular excitability by …

G-protein-gated inwardly rectifying K+(GIRK) channels are widely expressed in the brain
and are activated by at least eight different neurotransmitters. As K+ channels, they drive the …

Activation of the heteromeric G protein-gated inwardly rectifying K+ channel (GIRK) GIRK1
and GIRK4 subunits gives rise to I KACh, which controls excitability in atrial tissue. Although …

G protein-gated inwardly rectifying potassium (GIRK) channels are a family of K+-selective
ion channels that slow the firing rate of neurons and cardiac myocytes. GIRK channels are …

To investigate possible effects of adrenergic stimulation on G protein–activated inwardly
rectifying K+ channels (GIRK), acetylcholine (ACh)-evoked K+ current, IKACh, was recorded …