Object. The pathogenesis of delayed ischemic neurological deficits after subarachnoid
hemorrhage has been related to products of hemolysis. Topical brain superfusion of artificial …

OBJECTIVE: It has been proposed that delayed ischemic neurological deficits are induced
by red blood cell (RBC) products after subarachnoid hemorrhage. Prophylactic treatment …

Hemoglobin released from hemolysed erythrocytes has been postulated to be responsible
for delayed cerebral vasospasm after subarachnoid hemorrhage (SAH). However, the …

OBJECTIVE: To study the cerebral metabolism and its relationship to cerebral blood flow
(CBF) acutely after subarachnoid hemorrhage (SAH). METHODS: SAH was induced in rats …

Models have been devised and characterized in the laboratory rat for studying the
neuropathology of subarachnoid hemorrhage. Several ways of injecting blood via different …

OBJECTIVE: To study the occurrence of an inflammatory response in the brain after
subarachnoid hemorrhage and its relation to the decrease in acute cerebral blood flow …

OBJECTIVE: To investigate acute pathophysiological changes after subarachnoid
hemorrhage (SAH) in rats and compare endovascular perforation and double blood …

Object. The authors tested the hypotheses that subarachnoid hemorrhage (SAH) leads to
delayed cell death with the participation of apoptotic-like mechanisms and is influenced by …

Background and Purpose—Red blood cell (RBC) lysis contributes to brain edema formation
after intracerebral hemorrhage (ICH), and RBC hemolysate (oxyhemoglobin) has been …

Apoptosis is the term given to programmed cell death, which has been widely connected to
a number of intracranial pathologies including stroke, Alzheimer's disease, and more …