Serum concentrations of thyroid hormones tetraiodothyronine and triiodothyronine
commonly are low after head injury and brain death. Thyroid hormone replacement therapy …

Background Acute hypothyroidism after brain death results in hemodynamic impairments
that limit availability of donor hearts. Thyroid hormone infusions can halt that process and …

Experimental animal work has shown that thyroid hormone levels become undetectable 9
hours after brain death. It is unknown whether such an acutely hypothyroid state contributes …

Background Hormonal therapy to the brain-dead potential organ donor can include thyroid
hormone (triiodothyronine [T 3] or levothyroxine [T 4]), corticosteroids, antidiuretic hormone …

Objectives: To review all published clinical studies of thyroid hormone administration to
brain-dead potential organ donors. Methods: A search of PubMed using multiple search …

The aggressive management of brain‐dead (cadaveric) organ donors has been shown to
increase organs available for transplantation. Some centers use hormone therapy with …

Hormonal therapy to the brain-dead organ donor can include thyroid hormone
(triiodothyronine [T 3] or levothyroxine [T 4]), antidiuretic hormone, corticosteroids, or insulin …

Rationale: Brain death (BD) precipitates cardiac dysfunction impairing the ability to
transplant hearts from eligible organ donors. Retrospective studies have suggested that …

Triiodothyronine deficiency after brain death can result in progressive deterioration of
cardiac function in potential organ donors. We report on the use of triiodothyronine …

Background Brain death frequently induces hemodynamic instability and cardiac stunning.
Impairments in cardiac performance are major contributors to hearts from otherwise eligible …