MicroRNA-136-5p from endothelial progenitor cells-released extracellular vesicles mediates TXNIP to promote the dissolution of deep venous thrombosis

Y Feng, B Lei, H Zhang, L Niu, X Li, X Luo, F Zhang - Shock, 2022 - journals.lww.com
Objective: Endothelial progenitor cells-released extracellular vesicles (EPCs-EVs) have
previously been reported to promote the dissolution of deep venous thrombosis (DVT) …

MicroRNA-342-3p loaded by human umbilical cord mesenchymal stem cells-derived exosomes attenuates deep vein thrombosis by downregulating EDNRA

Z Pan, Q Chen, H Ding, H Li - Journal of Thrombosis and Thrombolysis, 2022 - Springer
Exosomes (exos) exert biological functions to maintain the dynamic balance of cells and
tissues by transferring biological cargo to recipient cells. Thus, this study explored human …

[HTML][HTML] Endothelial progenitor cell-derived exosomes, loaded with miR-126, promoted deep vein thrombosis resolution and recanalization

J Sun, Z Zhang, T Ma, Z Yang, J Zhang… - Stem cell research & …, 2018 - ncbi.nlm.nih.gov
Background Deep vein thrombosis (DVT) is caused by blood clotting in the deep veins.
Thrombosis resolution and recanalization can be accelerated by endothelial progenitor …

Upregulation of microRNA‐126 contributes to endothelial progenitor cell function in deep vein thrombosis via its target PIK3R2

Q Meng, W Wang, X Yu, W Li, L Kong… - Journal of Cellular …, 2015 - Wiley Online Library
Deep vein thrombosis (DVT) is a common complication of surgery. Endothelial progenitor
cells (EPCs) are recruited into resolving venous thrombi. In this report, we investigated the …

Downregulation of miR-125a-5p promotes endothelial progenitor cell migration and angiogenesis and alleviates deep vein thrombosis in mice via upregulation of …

J Yu, Y Jin, C Xu, C Fang, Z Zhang, L Chen… - Molecular Biotechnology, 2023 - Springer
Endothelial progenitor cells (EPCs) contribute to recanalization of deep vein thrombosis
(DVT). MicroRNAs (miRNAs) play regulatory roles in functions of EPCs, which is becoming a …

Upregulated miR‐206 aggravates deep vein thrombosis by regulating GJA1‐Mediated autophagy of endothelial progenitor cells

Y Li, J Ge, Y Yin, R Yang, J Kong… - Cardiovascular …, 2022 - Wiley Online Library
Background. Deep vein thrombosis (DVT) is the third most prevalent vascular disease
worldwide. MicroRNAs (miRNAs) play regulatory roles in functions of endothelial progenitor …

Down-regulation of miR-361-5p promotes the viability, migration and tube formation of endothelial progenitor cells via targeting FGF1

X Yang, Y Song, Y Sun, M Wang, Y Xiang - Bioscience reports, 2020 - portlandpress.com
Transplantion of bone marrow-derived endothelial progenitor cells (EPCs) may be a novel
treatment for deep venous thrombosis (DVT). The present study probed into the role of …

MicroRNA-195 regulates proliferation, migration, angiogenesis and autophagy of endothelial progenitor cells by targeting GABARAPL1

J Mo, D Zhang, R Yang - Bioscience reports, 2016 - portlandpress.com
Deep vein thrombosis (DVT) is a common type of venous thrombosis. Successful resolution
of DVT-related thrombi is important in the treatment of DVT. Endothelial progenitor cells …

[HTML][HTML] miR-150 regulates endothelial progenitor cell differentiation via Akt and promotes thrombus resolution

X Du, N Hu, H Yu, L Hong, F Ran, D Huang… - Stem Cell Research & …, 2020 - Springer
Background Deep venous thrombosis (DVT) constitutes a major global disease burden.
Endothelial progenitor cells (EPCs) have been described in association with recanalization …

MiR-143-3p targets ATG2B to inhibit autophagy and promote endothelial progenitor cells tube formation in deep vein thrombosis

W Zhang, P Chen, H Zong, Y Ding, R Yan - Tissue and Cell, 2020 - Elsevier
Deep vein thrombosis (DVT) is a common disease in vascular surgery. In recent study,
microRNA (miRNA) plays a regulatory role in function of Endothelial progenitor cells (EPCs) …