Background Neuroinflammation and blood-brain barrier (BBB) disruption are two vital
mechanisms of secondary brain injury following intracerebral hemorrhage (ICH). Recently …

Background Neuroinflammation plays an important role in the pathogenesis of intracerebral
hemorrhage (ICH)-induced secondary brain injury. Activation of melanocortin receptor 4 …

Oxidative stress and neuronal apoptosis play crucial roles in secondary brain injury (SBI)
after intracerebral hemorrhage (ICH). Recently, Nle4‐D‐Phe7‐α‐melanocyte‐stimulating …

Neuroinflammation plays a vital role in early brain injury (EBI) following subarachnoid
hemorrhage (SAH). The hypothesis of this study was that activation of melanocortin 1 …

Background Microglia-mediated neuroinflammation plays a crucial role in the pathogenesis
of hypoxic-ischemic (HI)-induced brain injury. Activation of melanocortin-1 receptor (MC1R) …

Neuroinflammation is an essential mechanism involved in the pathogenesis of
subarachnoid hemorrhage (SAH)-induced brain injury. Recently, Netrin-1 (NTN-1) is well …

In inflammation-associated progressive neuroinflammatory disorders, such as multiple
sclerosis (MS), inflammatory infiltrates containing T helper 1 (TH1) and TH17 cells cause …

Nucleotide oligomerization domain (NOD)-like receptor protein-3 (NLRP3) inflammasome is
a multiprotein complex, which results in the inflammatory response in early brain injury (EBI) …

The activation of CC chemokine receptor type 1 (CCR1) has been shown to be pro-
inflammatory in several animal models of neurological diseases. The objective of this study …

Neuroinflammation significantly contributes to brain injury and neurological deterioration
following intracerebral hemorrhage (ICH). MicroRNA-152 (miR-152) was reported to be …