Background: Current obesity theories suggest that the repeated intake of highly palatable
high-sugar foods causes adaptions in the striatum, parietal lobe, and prefrontal and visual …

Artificially sweetened beverage consumption has been linked to obesity, and it has been
hypothesized that considerable exposure to nonnutritive sweeteners may be associated with …

Background: Although the intake of high-fat and high-sugar food activates mesolimbic
reward, gustatory, and oral somatosensory brain regions, contributing to overeating, few …

There is a general consensus that overconsumption of sugar-sweetened beverages
contributes to the prevalence of obesity and related comorbidities such as type 2 diabetes …

Western diets rich in fat and sugar promote excess calorie intake and weight gain; however,
the underlying mechanisms are unclear. Despite a well-documented association between …

Background Ingestion of sweet food is driven by central reward circuits and restrained by
endocrine and neurocrine satiety signals. The specific influence of sucrose intake on central …

After food consumption, the motivation to eat (wanting) decreases and associated brain
reward responses change. Wanting-related brain responses and how these are affected by …

Abstract Purpose of Review The mechanistic role of sugar-sweetened beverage (SSB) in the
etiology of obesity is undetermined. We address whether, compared to other foods, does …

Objective Consuming sweet foods, even when sated, can lead to unwanted weight gain.
Contextual factors, such as longer time fasting, subjective hunger, and body mass index …

Background: The consumption of fat is regulated by reward and homeostatic pathways, but
no studies to our knowledge have examined the role of high-fat meal (HFM) intake on …