Active Helicobacter pylori-associated gastritis is characterized by a dense mucosal
infiltration with granulocytes. Since H. pylori is noninvasive, secondary signals must induce …

Accumulating evidence suggests an important role of interleukin-8 (IL-8) in Helicobacter
pylori infection-associated chronic atrophic gastritis and peptic ulcer. We observed in this …

Gastric infection with Helicobacter pylori activates a mucosal inflammatory response by
mononuclear cells and neutrophils that includes expression of cytokines interleukin-1 beta …

Following exposure to Helicobacter pylori cells, epithelial cell lines secreted interleukin-6 (IL-
6) and IL-8 but not tumor necrosis factor alpha. Purified IL-6 alone did not stimulate IL-8 …

Background/Aims: Helicobacter pylori is associated with neutrophil infiltrates, although the
mechanism of their recruitment is only partially defined. The aim of the study was to …

When Helicobacter pylori arrives in the human stomach, it may penetrate the mucin layer
and adhere to the gastric epithelial cells or it may pass through the stomach without …

Background: The colonization of the gastric mucosa with Helicobacter pylori is accompanied
by elevated levels of proinflammatory cytokines, such as interleukin‐1 (IL‐1), IL‐6, and IL‐8 …

The gastroduodenal response to chronic Helicobacter pylori infection is characterized by the
infiltration of plasma cells, lymphocytes, neutrophils and monocytes into the mucosa …

Despite the fact that Helicobacter pylori is known to be non‐invasive, mucosal infiltration of
inflammatory cells have been observed in the gastric mucosa. The exact pathogenesis of …

Objective: To review the role of interleukin (IL)-8 in the immunopathology of Helicobacter
pylori-associated gastroduodenal disease. Method: Literature review. Results: In H. pylori …