Background: Excessive fructose intake causes metabolic syndrome in animals and can be
partially prevented by lowering the uric acid level. We tested the hypothesis that fructose …

The worldwide epidemic of metabolic syndrome correlates with an elevation in serum uric
acid as well as a marked increase in total fructose intake (in the form of table sugar and high …

With the widespread popularity of hyperuricemia, it has become a severe threat to human
public health. Accumulating evidence suggests that dietary fructose has a close relationship …

Although fructose consumption has dramatically increased and is suspected to be causally
linked to metabolic abnormalities, the mechanisms involved are still only partially …

We are currently in the midst of an epidemic of metabolic disorders, which may, in part, be
explained by excess fructose intake. This theory is supported by epidemiological …

Fructose-induced hyperuricemia might have a causal role in metabolic syndrome,
hypertension, and other chronic disease. However, no study has investigated whether sugar …

The association between fructose and increased blood pressure is still incompletely defined,
because experimental studies have produced dissimilar conclusions. Amplified vasopressor …

There is a direct relationship between fructose intake and serum levels of uric acid (UA),
which is the final product of purine metabolism. Recent preclinical and clinical evidence …

We propose that excessive fructose intake (> 50 g/d) may be one of the underlying etiologies
of metabolic syndrome and type 2 diabetes. The primary sources of fructose are sugar …

Background Prospective studies in humans examining the effects of fructose consumption
on biological markers associated with the development of metabolic syndrome are lacking …