[HTML][HTML] Inhibition of pulmonary fibrosis in mice by CXCL10 requires glycosaminoglycan binding and syndecan-4

D Jiang, J Liang, GS Campanella… - The Journal of …, 2010 - Am Soc Clin Investig
Pulmonary fibrosis is a progressive, dysregulated response to injury culminating in
compromised lung function due to excess extracellular matrix production. The heparan …

Inhibition of pulmonary fibrosis in mice by CXCL10 requires glycosaminoglycan binding and syndecan-4.

D Jiang, J Liang, GS Campanella, R Guo… - The Journal of …, 2010 - europepmc.org
Pulmonary fibrosis is a progressive, dysregulated response to injury culminating in
compromised lung function due to excess extracellular matrix production. The heparan …

[PDF][PDF] Inhibition of pulmonary fibrosis in mice by CXCL10 requires glycosaminoglycan binding and syndecan-4

D Jiang, J Liang, GS Campanella, R Guo, S Yu, T Xie… - researchgate.net
Pulmonary fibrosis is a progressive, dysregulated response to injury culminating in
compromised lung function due to excess extracellular matrix production. The heparan …

[PDF][PDF] Inhibition of pulmonary fibrosis in mice by CXCL10 requires glycosaminoglycan binding and syndecan-4

D Jiang, J Liang, GS Campanella, R Guo, S Yu, T Xie… - academia.edu
Pulmonary fibrosis is a progressive, dysregulated response to injury culminating in
compromised lung function due to excess extracellular matrix production. The heparan …

[HTML][HTML] Inhibition of pulmonary fibrosis in mice by CXCL10 requires glycosaminoglycan binding and syndecan-4

D Jiang, J Liang, GS Campanella, R Guo… - The Journal of …, 2010 - ncbi.nlm.nih.gov
Pulmonary fibrosis is a progressive, dysregulated response to injury culminating in
compromised lung function due to excess extracellular matrix production. The heparan …

Inhibition of pulmonary fibrosis in mice by CXCL10 requires glycosaminoglycan binding and syndecan-4

D Jiang, J Liang, GS Campanella… - The Journal of …, 2010 - pubmed.ncbi.nlm.nih.gov
Pulmonary fibrosis is a progressive, dysregulated response to injury culminating in
compromised lung function due to excess extracellular matrix production. The heparan …

[引用][C] Inhibition of pulmonary fibrosis in mice by CXCL10 requires glycosaminoglycan binding and syndecan-4

D Jiang, J Liang, GS Campanella, R Guo, S Yu… - Journal of Clinical …, 2010 - cir.nii.ac.jp
Inhibition of pulmonary fibrosis in mice by CXCL10 requires glycosaminoglycan binding and
syndecan-4 | CiNii Research CiNii 国立情報学研究所 学術情報ナビゲータ[サイニィ] 詳細へ移動 …

Inhibition of pulmonary fibrosis in mice by CXCL10 requires glycosaminoglycan binding and syndecan-4

D Jiang, J Liang, GS Campanella… - Journal of Clinical …, 2010 - search.proquest.com
Pulmonary fibrosis is a progressive, dysregulated response to injury culminating in
compromised lung function due to excess extracellular matrix production. The heparan …

[PDF][PDF] Inhibition of pulmonary fibrosis in mice by CXCL10 requires glycosaminoglycan binding and syndecan-4

D Jiang, J Liang, GS Campanella, R Guo, S Yu, T Xie… - dukespace.lib.duke.edu
Pulmonary fibrosis is a progressive, dysregulated response to injury culminating in
compromised lung function due to excess extracellular matrix production. The heparan …

[PDF][PDF] Inhibition of pulmonary fibrosis in mice by CXCL10 requires glycosaminoglycan binding and syndecan-4

D Jiang, J Liang, GS Campanella, R Guo, S Yu, T Xie… - Am Soc Clin Investig
Pulmonary fibrosis is a progressive, dysregulated response to injury culminating in
compromised lung function due to excess extracellular matrix production. The heparan …