Understanding vascular inflammation and myofibroblast crosstalk is critical to developing
therapies for fibrotic diseases. Here we report the development of a novel human Tendon-on …

Understanding vascular inflammation and myofibroblast crosstalk is critical to developing
therapies for fibrotic diseases. Here we report the development of a novel human Tendon-on …

Vascular inflammation and activation of myofibroblasts are significant contributors to the
progression of fibrosis, which can severely impair tissue function. In various tissues …

Tendinopathies are poorly understood diseases for which treatment remains challenging.
Relevant in vitro models to study human tendon physiology and pathophysiology are …

Tendons are among the most mechanically stressed tissues of the body, with a functional
core of type‐I collagen fibers maintained by embedded stromal fibroblasts known as …

Tendons enable locomotion by transferring muscle forces to bones. They rely on a tough
tendon core comprising collagen fibers and stromal cell populations. This load-bearing core …

Tendinopathy is a multi-faceted pathology characterized by alterations in tendon
microstructure, cellularity and collagen composition. Challenged by the possibility of …

Tendons enable locomotion by transferring muscle forces to bones. They rely on a tough
tendon core comprising collagen fibers and stromal cell populations. This load-bearing core …

Fibrosis, a hallmark of many cardiac and pulmonary diseases, is characterized by excess
deposition of extracellular matrix proteins and increased tissue stiffness. This serious …

Acute tendon injuries represent a major clinical burden, due in part to tendon's fibrotic
healing process. Complete restoration of functional properties is prevented by excessive …