The HIV-1 vpr protein acts as a negative regulator of apoptosis in a human lymphoblastoid T cell line: possible implications for the pathogenesis of AIDS

L Conti, G Rainaldi, P Matarrese, B Varano… - The Journal of …, 1998 - rupress.org
Although apoptosis is considered one of the major mechanisms of CD4+ T cell depletion in
HIV-infected patients, the virus-infected cells somehow appear to be protected from …

[PDF][PDF] The HIV-1 vpr Protein Acts as a Negative Regulator of Apoptosis in a Human Lymphoblastoid T Cell Line: Possible Implications for the Pathogenesis of AIDS

L Conti, G Rainaldi, P Matarrese, B Varano… - pdfs.semanticscholar.org
Although apoptosis is considered one of the major mechanisms of CD4 T cell depletion in
HIV-infected patients, the virus-infected cells somehow appear to be protected from …

The HIV-1 vpr Protein Acts as a Negative Regulator of Apoptosis in a Human Lymphoblastoid T Cell Line: Possible Implications for the Pathogenesis of AIDS

L Conti, G Rainaldi, P Matarrese, B Varano… - The Journal of …, 1998 - cir.nii.ac.jp
抄録< jats: p> Although apoptosis is considered one of the major mechanisms of CD4+ T cell
depletion in HIV-infected patients, the virus-infected cells somehow appear to be protected …

The HIV-1 vpr protein acts as a negative regulator of apoptosis in a human lymphoblastoid T cell line: possible implications for the pathogenesis of AIDS.

L Conti, G Rainaldi, P Matarrese, B Varano… - The Journal of …, 1998 - europepmc.org
Although apoptosis is considered one of the major mechanisms of CD4+ T cell depletion in
HIV-infected patients, the virus-infected cells somehow appear to be protected from …

[PDF][PDF] The HIV-1 vpr Protein Acts as a Negative Regulator of Apoptosis in a Human Lymphoblastoid T Cell Line: Possible Implications for the Pathogenesis of AIDS

L Conti, G Rainaldi, P Matarrese, B Varano… - researchgate.net
Although apoptosis is considered one of the major mechanisms of CD4 T cell depletion in
HIV-infected patients, the virus-infected cells somehow appear to be protected from …

The HIV-1 vpr protein acts as a negative regulator of apoptosis in a human lymphoblastoid T cell line: possible implications for the pathogenesis of AIDS

L Conti, G Rainaldi, P Matarrese… - The Journal of …, 1998 - pubmed.ncbi.nlm.nih.gov
Although apoptosis is considered one of the major mechanisms of CD4 (+) T cell depletion
in HIV-infected patients, the virus-infected cells somehow appear to be protected from …

The HIV-1 Vpr protein acts as a negative regulator of apoptosis in a human lymphoblastoid T cell line: possible implications for the pathogenesis of AIDS.

L Conti, G Rainaldi, P Matarrese, B Varano… - 1998 - cabidigitallibrary.org
This study showed that the constitutive expression of the HIV-1 Vpr protein results in a
marked inhibition of apoptosis induced by different stimuli, such as a combination of …

[HTML][HTML] The HIV-1 vpr Protein Acts as a Negative Regulator of Apoptosis in a Human Lymphoblastoid T Cell Line: Possible Implications for the Pathogenesis of AIDS

L Conti, G Rainaldi, P Matarrese, B Varano… - The Journal of …, 1998 - ncbi.nlm.nih.gov
Although apoptosis is considered one of the major mechanisms of CD4+ T cell depletion in
HIV-infected patients, the virus-infected cells somehow appear to be protected from …

[PDF][PDF] The HIV-1 vpr Protein Acts as a Negative Regulator of Apoptosis in a Human Lymphoblastoid T Cell Line: Possible Implications for the Pathogenesis of AIDS

L Conti, S Columba, A Sato, F Belardelli, W Malorni… - academia.edu
Although apoptosis is considered one of the major mechanisms of CD4 T cell depletion in
HIV-infected patients, the virus-infected cells somehow appear to be protected from …

[PDF][PDF] The HIV-1 vpr Protein Acts as a Negative Regulator of Apoptosis in a Human Lymphoblastoid T Cell Line: Possible Implications for the Pathogenesis of AIDS

L Conti, G Rainaldi, P Matarrese, B Varano… - Citeseer
Although apoptosis is considered one of the major mechanisms of CD4 T cell depletion in
HIV-infected patients, the virus-infected cells somehow appear to be protected from …