Binding mechanisms of amyloid-like peptides to lipid bilayers and effects of divalent cations

Y Yang, S Jalali, BL Nilsson… - ACS Chemical …, 2021 - ACS Publications
In several neurodegenerative diseases, cell toxicity can emerge from damage produced by
amyloid aggregates to lipid membranes. The details accounting for this damage are poorly …
被引用次数：21 相关文章
[PDF] nsf.gov

[PDF][PDF] Binding Mechanisms of Amyloid-like Peptides to Lipid Bilayers and Effects of Divalent Cations

Y Yang, S Jalali, BL Nilsson, CL Dias - ACS Chemical Neuroscience, 2021 - par.nsf.gov
In several neurodegenerative diseases, cell toxicity can emerge from damage produced by
amyloid aggregates to lipid membranes. The details accounting for this damage are poorly …

Binding Mechanisms of Amyloid-like Peptides to Lipid Bilayers and Effects of Divalent Cations.

Y Yang, S Jalali, BL Nilsson, CL Dias - ACS Chemical Neuroscience, 2021 - europepmc.org
In several neurodegenerative diseases, cell toxicity can emerge from damage produced by
amyloid aggregates to lipid membranes. The details accounting for this damage are poorly …

Binding Mechanisms of Amyloid-like Peptides to Lipid Bilayers and Effects of Divalent Cations

Y Yang, S Jalali, BL Nilsson… - ACS chemical …, 2021 - pubmed.ncbi.nlm.nih.gov
In several neurodegenerative diseases, cell toxicity can emerge from damage produced by
amyloid aggregates to lipid membranes. The details accounting for this damage are poorly …

Binding Mechanisms of Amyloid-like Peptides to Lipid Bilayers and Effects of Divalent Cations

Y Yang, S Jalali, BL Nilsson… - ACS Chemical …, 2021 - researchwith.njit.edu
In several neurodegenerative diseases, cell toxicity can emerge from damage produced by
amyloid aggregates to lipid membranes. The details accounting for this damage are poorly …
[PDF] njit.edu

[PDF][PDF] Binding Mechanisms of Amyloid-like Peptides to Lipid Bilayers and Effects of Divalent Cations

Y Yang, S Jalali, BL Nilsson, CL Dias - 2021 - njit.edu
In several neurodegenerative diseases, cell toxicity can emerge from damage produced by
amyloid aggregates to lipid membranes. The details accounting for this damage are poorly …