Helicobacter pylori cag+ strains enhance gastric epithelial cell proliferation and attenuate
apoptosis in vivo, which may partially explain the increased risk of gastric cancer associated …

Helicobacter pylori colonization induces inflammation in essentially all hosts, a persistent
process that increases the risk of developing distal gastric adenocarcinoma. However, only …

Infection with the bacterium Helicobacter pylori is associated epidemiologically with
development of gastric cancer. To better understand the role of H. pylori in carcinogenesis …

Abstract Background: Infection with Helicobacter pylori induces chronic gastritis in virtually
all infected persons, and such gastritis has been associated with an increased risk of …

Background & Aims: Human colonization with Helicobacter pylori increases the risk for distal
gastric adenocarcinoma, possibly by altering gastric epithelial cell cycle events and/or …

Despite the fact that the association ofHelicobacterpylori with an increased risk of
gastriccancer is well documented, the exact mechanisms of thisassociation have not been …

Gastric colonization by Helicobacter pylori is a risk factor for noncardia gastric cancer. The
association between H. pylori and cancer may be attributable to increased epithelial cell …

Helicobacter pylori is the strongest known risk factor for gastric adenocarcinoma, and strains
that possess the cag secretion system, which translocates the bacterial effector CagA into …

Infection with Helicobacter pylori (H. pylori) increases stomach cancer risk. Helicobacter
pylori strains with the cag pathogenicity island (PAI) induce more severe inflammation in the …

Adenocarcinoma of the stomach is the second most common cause of cancer mortality in the
world. The purpose of this study was to evaluate the potential role in carcinogenesis of two …