Most drugs of abuse easily cross the placenta and can affect fetal brain development. In
utero exposures to drugs thus can have long-lasting implications for brain structure and …

Adverse in utero events can alter the development and function of numerous physiological
systems, giving rise to lasting neurodevelopmental deficits. In particular, data have shown …

Aim: Prenatal alcohol exposure (PAE) can cause fetal alcohol spectrum disorders (FASD).
Previously, we assessed PAE in brain tissue from mouse models, however whether these …

Prenatal ethanol exposure (PEE)(mainly through maternal alcohol consumption) has
become widespread. However, studies suggest that it can cause intrauterine growth …

Alcohol consumption during pregnancy and its damaging consequences on the developing
infant brain are significant public health, social, and economic issues. The major distinctive …

Alcohol consumption and its abuse is a major health problem resulting in significant
healthcare cost in the United States. Chronic alcoholism results in damage to most of the …

There is abundant evidence that prenatal alcohol exposure leads to a range of behavioral
and cognitive impairments, categorized under the term fetal alcohol spectrum disorders …

Prenatal alcohol (ethanol) exposure (PAE) is the underlying cause for a variety of birth
defects and neurodevelopmental deficits referred to as “Fetal Alcohol Spectrum Disorders …

Brain development in mammals is long lasting. It begins early during embryonic growth and
is finalized in early adulthood. This progression represents a delicate choreography of …

Background Prenatal alcohol exposure is recognized for altering DNA methylation profiles of
brain cells during development, and to be part of the molecular basis underpinning Fetal …