[PDF][PDF] Bile acid conjugation deficiency causes hypercholanemia, hyperphagia, islet dysfunction, and gut dysbiosis in mice

BD Alrehaili, M Lee, S Takahashi… - Hepatology …, 2022 - Wiley Online Library
Bile acid‐CoA: amino acid N‐acyltransferase (BAAT) catalyzes bile acid conjugation, the
last step in bile acid synthesis. BAAT gene mutation in humans results in hypercholanemia …

Bile acid conjugation deficiency causes hypercholanemia, hyperphagia, islet dysfunction, and gut dysbiosis in mice

BD Alrehaili, M Lee, S Takahashi, R Novak… - Hepatology …, 2022 - pure.psu.edu
Bile acid-CoA: amino acid N-acyltransferase (BAAT) catalyzes bile acid conjugation, the last
step in bile acid synthesis. BAAT gene mutation in humans results in hypercholanemia …

Bile acid conjugation deficiency causes hypercholanemia, hyperphagia, islet dysfunction, and gut dysbiosis in mice

BD Alrehaili, M Lee, S Takahashi… - Hepatology …, 2022 - experts.nebraska.edu
Bile acid-CoA: amino acid N-acyltransferase (BAAT) catalyzes bile acid conjugation, the last
step in bile acid synthesis. BAAT gene mutation in humans results in hypercholanemia …

[HTML][HTML] Bile acid conjugation deficiency causes hypercholanemia, hyperphagia, islet dysfunction, and gut dysbiosis in mice

BD Alrehaili, M Lee, S Takahashi, R Novak… - Hepatology …, 2022 - ncbi.nlm.nih.gov
Bile acid‐CoA: amino acid N‐acyltransferase (BAAT) catalyzes bile acid conjugation, the
last step in bile acid synthesis. BAAT gene mutation in humans results in hypercholanemia …

Bile acid conjugation deficiency causes hypercholanemia, hyperphagia, islet dysfunction, and gut dysbiosis in mice

BD Alrehaili, M Lee, S Takahashi… - Hepatology …, 2022 - pubmed.ncbi.nlm.nih.gov
Bile acid-CoA: amino acid N-acyltransferase (BAAT) catalyzes bile acid conjugation, the last
step in bile acid synthesis. BAAT gene mutation in humans results in hypercholanemia …

Bile acid conjugation deficiency causes hypercholanemia, hyperphagia, islet dysfunction, and gut dysbiosis in mice.

BD Alrehaili, M Lee, S Takahashi, R Novak… - Hepatology …, 2022 - europepmc.org
Bile acid‐CoA: amino acid N‐acyltransferase (BAAT) catalyzes bile acid conjugation, the
last step in bile acid synthesis. BAAT gene mutation in humans results in hypercholanemia …

Bile acid conjugation deficiency causes hypercholanemia, hyperphagia, islet dysfunction, and gut dysbiosis in mice

BD Alrehaili, M Lee, S Takahashi… - Hepatology …, 2022 - search.proquest.com
Bile acid‐CoA: amino acid N‐acyltransferase (BAAT) catalyzes bile acid conjugation, the
last step in bile acid synthesis. BAAT gene mutation in humans results in hypercholanemia …

[HTML][HTML] Bile acid conjugation deficiency causes hypercholanemia, hyperphagia, islet dysfunction, and gut dysbiosis in mice

BD Alrehaili, M Lee, S Takahashi… - Hepatology …, 2022 - journals.lww.com
Bile acid‐CoA: amino acid N‐acyltransferase (BAAT) catalyzes bile acid conjugation, the
last step in bile acid synthesis. BAAT gene mutation in humans results in hypercholanemia …

Bile acid conjugation deficiency causes hypercholanemia, hyperphagia, islet dysfunction, and gut dysbiosis in mice.

BD Alrehaili, M Lee, S Takahashi, R Novak… - Hepatology …, 2022 - europepmc.org
Bile acid‐CoA: amino acid N‐acyltransferase (BAAT) catalyzes bile acid conjugation, the
last step in bile acid synthesis. BAAT gene mutation in humans results in hypercholanemia …