AG–> A transition at-78 base pairs from the transcription start site of the apolipoprotein AI
(apoA-I) gene has been associated with increased apoA-I serum levels in humans. We …

The plasma lipid response to changes in dietary fat and cholesterol can vary between
individuals. At present, responders cannot be identified in advance. An adenine to guanine …

The effect associated with the substitution of adenine (A) for guanidine (G) in the promoter
region of the apolipoprotein AI gene (− 75 bp) with plasma apo AI and high‐density …

The influence of the guanidine to adenine (G/A) substitution in the promoter region of the
apolipoprotein (apo) AI gene (at− 75 bp) on serum lipids and apolipoproteins was studied in …

Expression of the apolipoprotein AI (apoAI) gene in the liver is controlled by a liver-specific
enhancer. The function of this enhancer depends on synergistic interactions between …

Liver-specific expression of the apolipoprotein AI (apoA-I) gene is controlled by the
coordinate action of transcription factors bound to three sites (A, B, and C) located within a …

We herein demonstrate competence of the 5′ upstream region− 1374 to+ 16 of the human
growth factor-activatable Na+/H+ exchanger (NHE-1) gene to promote transcription of the …

Apolipoprotein AI (apoA-I) gene expression is known to be regulated by nutritional and
hormonal factors. Experiments were conducted to determine the effects of vitamin A …

We examined the impact of a G→ A mutation at position-75 of the apolipoprotein AI gene
promoter in subjects with hypertriglyceridaemia from two racial groups, Caucasians (n= 52) …

Abstract Two cupruretic chelators, 2, 3, 2-tetramine and diamsar were used to deplete
copper from Hep G2 cells. Studies using incremental concentrations of chelators, an …