Ischemia-reperfusion injury is, at least in part, responsible for the morbidity associated with
liver surgery under total vascular exclusion or after liver transplantation. The …

Warm and cold hepatic ischemia followed by reperfusion leads to necrotic cell death
(oncosis), which often occurs within minutes of reperfusion. Recent studies also suggest a …

This review highlights recent advances in our understanding of mechanisms underlying
reperfusion injury to the liver after warm hepatic ischemia. Sinusoidal endothelial cells and …

This article summarizes strategies to protect the liver from injuries caused by ischemia and
reperfusion. Three different sections (ie, surgical and pharmacologic strategies and gene …

Background/Aims: Ischemic preconditioning (IP) is known to protect hepatic tissue from
ischemia–reperfusion injury. However, the mechanisms involved are not fully understood …

Ischemia and reperfusion (IR) injury of the liver consists of two distinct phases. The first
phase is caused by acute cellular injury at 3 to 6 h postreperfusion, which may be mainly …

Ischemia–reperfusion injury is the main cause of primary dysfunction or nonfunction after
liver transplantation. In addition, the liver suffers from warm ischemia–reperfusion injury …

Platelets have been implicated in the pathophysiology of ischemia–reperfusion (I/R) injury.
This study was designed to analyze platelet–endothelial cell interactions after I/R of the liver …

Ischemia/reperfusion injury of the liver has two distinct phases that contribute to hepatocyte
damage. The acute phase is characterized by Kupffer cell production of reactive oxygen …

Background: Hepatic ischemia/reperfusion (I/R) injury is characterized by features such as
cholestasis. Mitochondria become susceptible to damage during ischemia and after …