The neurologist’s dilemma: A comprehensive clinical review of Bell’s palsy, with emphasis on
current management trends - PMC Back to Top Skip to main content NIH NLM Logo Access keys …

Primary infection of humans with varicella zoster virus (VZV) causes varicella (chickenpox),
after which the virus becomes latent in cranial nerve ganglia, dorsal root ganglia and …

Objectives: Stroke and TIA are recognized complications of acute herpes zoster (HZ). In this
study, we evaluated HZ as a risk factor for cerebrovascular disease (stroke and TIA) and …

Adults developing zoster are at increased risk of stroke for 6 months with a> 3-fold increased
risk following zoster ophthalmicus. Incidence ratios of stroke were lower among those …

Most infectious pathogens have anecdotal evidence to support a link with stroke, but certain
pathogens have more robust associations, in which causation is probable. Few dedicated …

Primary infection with varicella-zoster virus (VZV), an exclusively human neurotropic α-
herpesvirus, causes varicella (chickenpox), after which virus becomes latent in ganglionic …

Varicella zoster virus (VZV) vasculopathy is caused by productive virus infection of cerebral
arteries, leading to inflammation, pathological vascular remodeling, and ischemic or …

Objective Herpes zoster results from reactivation of the endogenous varicella zoster virus
infection. Previous studies have shown that herpes zoster and postherpetic neuralgia were …

Background The varicella zoster virus (VZV) is a highly neurotropic virus that, after the
primary infection, remains latent in the nerve cells and can reactivate many years later …

Inflammation and infection have been noted to increase stroke risk. However, the
association between septicaemia and increased risk of stroke remains unclear. This …