Controlling DNA replication origins in response to DNA damage–inhibit globally, activate locally

M Yekezare, B Gómez-González… - Journal of cell …, 2013 - journals.biologists.com
DNA replication in eukaryotic cells initiates from multiple replication origins that are
distributed throughout the genome. Coordinating the usage of these origins is crucial to …

Replication stress caused by low MCM expression limits fetal erythropoiesis and hematopoietic stem cell functionality

S Alvarez, M Díaz, J Flach, S Rodriguez-Acebes… - Nature …, 2015 - nature.com
Replicative stress during embryonic development influences ageing and predisposition to
disease in adults. A protective mechanism against replicative stress is provided by the …

Polε instability drives replication stress, abnormal development, and tumorigenesis

R Bellelli, V Borel, C Logan, J Svendsen, DE Cox… - Molecular cell, 2018 - cell.com
DNA polymerase ε (POLE) is a four-subunit complex and the major leading strand
polymerase in eukaryotes. Budding yeast orthologs of POLE3 and POLE4 promote Polε …

Convergence of SIRT1 and ATR signaling to modulate replication origin dormancy

BL Thakur, AM Baris, H Fu, CE Redon… - Nucleic acids …, 2022 - academic.oup.com
During routine genome duplication, many potential replication origins remain inactive or
'dormant'. Such origin dormancy is achieved, in part, by an interaction with the metabolic …

Dormant origins as a built-in safeguard in eukaryotic DNA replication against genome instability and disease development

N Shima, KD Pederson - DNA repair, 2017 - Elsevier
DNA replication is a prerequisite for cell proliferation, yet it can be increasingly challenging
for a eukaryotic cell to faithfully duplicate its genome as its size and complexity expands …

Comparative Oncogenomics Implicates the Neurofibromin 1 Gene (NF1) as a Breast Cancer Driver

MD Wallace, AD Pfefferle, L Shen, AJ McNairn… - Genetics, 2012 - academic.oup.com
Identifying genomic alterations driving breast cancer is complicated by tumor diversity and
genetic heterogeneity. Relevant mouse models are powerful for untangling this problem …

Chronic DNA replication stress reduces replicative lifespan of cells by TRP53-dependent, microRNA-assisted MCM2-7 downregulation

G Bai, MB Smolka, JC Schimenti - PLoS Genetics, 2016 - journals.plos.org
Circumstances that compromise efficient DNA replication, such as disruptions to replication
fork progression, cause a state known as DNA replication stress (RS). Whereas normally …

Post-transcriptional homeostasis and regulation of MCM2–7 in mammalian cells

CH Chuang, D Yang, G Bai, A Freeland… - Nucleic acids …, 2012 - academic.oup.com
Abstract The MiniChromosome Maintenance 2-7 (MCM2-7) complex provides essential
replicative helicase function. Insufficient MCMs impair the cell cycle and cause genomic …

[HTML][HTML] Mcm2 hypomorph leads to acute leukemia or hematopoietic stem cell failure, dependent on genetic context

T Matsukawa, M Yin, T Baslan, YJ Chung… - … : official publication of …, 2022 - ncbi.nlm.nih.gov
Abstract Minichromosome maintenance proteins (Mcm2–7) form a hexameric complex that
unwinds DNA ahead of a replicative fork. Deficiency of Mcm proteins leads to replicative …

A concomitant loss of dormant origins and FANCC exacerbates genome instability by impairing DNA replication fork progression

SW Luebben, T Kawabata, CS Johnson… - Nucleic acids …, 2014 - academic.oup.com
Accumulating evidence suggests that dormant DNA replication origins play an important role
in the recovery of stalled forks. However, their functional interactions with other fork recovery …