Adequate placentation, placental tissue remodeling and vascularization is essential for the
success of gestation and optimal fetal growth. Recently, it was suggested that abnormal …

Pregnancy is a unique condition, and the vascular processes that are required for this
undertaking are both complex and extensive. In this review, we discuss the vascular …

Background Prenatal alcohol exposure is the most common cause of birth defects and
intellectual disabilities and can increase the risk of stillbirth and negatively impact fetal …

Introduction Prenatal ethanol exposure compromises fetal growth by impairing placentation.
Invasive trophoblastic cells, which mediate placentation, express the insulin-IGF regulated …

It is known that inhibiting 11β-hydroxysteroid dehydrogenase type 2 (11β-HSD2) expression
in the placenta can cause fetal over-exposure to maternal glucocorticoids and induce …

Background Fetal alcohol spectrum disorders (FASD) are preventable adverse outcomes
consequent to prenatal alcohol exposure. Supplemental choline confers neuroprotection to …

Significance: Prenatal exposure to ethanol causes several morphological and
neurobehavioral deficits. While there are some studies on the effects of ethanol exposure on …

(1) Background: Programming of atherosclerosis results in vascular structure and function
alterations, which may be attributed to fetal exposure to maternal tobacco smoking, alcohol …

Fetal alcohol spectrum disorders (FASD) describe neurodevelopmental deficits in children
exposed to alcohol in utero. We hypothesized that gestational alcohol significantly alters …

Prenatal alcohol exposure is known to cause fetal growth restriction and disturbances in
amino acid bioavailability. Alterations in these parameters can persist into adulthood and …