Intracerebral hemorrhage (ICH) is a subtype of stroke with high mortality and disability but
no specific or effective treatment. In the last two decades, much has been learned about the …

The method of iron-dependent cell death known as ferroptosis is distinct from apoptosis. The
suppression of ferroptosis after intracerebral hemorrhage (ICH) will effectively treat ICH and …

Intracerebral hemorrhage (ICH) induces a strong hematoma-related neuroinflammatory
reaction and alters peripheral immune homeostasis. Recent research has found that gut …

Background: Promotion of hematoma resolution in a timely manner reduces intracerebral
hemorrhage (ICH) brain injury induced by toxic blood components and subsequent …

Aims Blood‐borne monocytes/macrophages infiltrate the brain in massive numbers after
ischemic stroke, but their impact on poststroke brain injury and recovery remains elusive …

Background and Purpose—Enhancement of erythrophagocytosis by macrophages in a
timely manner can limit the toxic effects of erythrocyte metabolites and promote brain …

Background and Purpose—Our previous studies found that erythrocyte CD47 has a role in
regulating hematoma resolution following experimental intracerebral hemorrhage (ICH). The …

Polarization of microglia/macrophages toward the pro-inflammatory phenotype is an
important contributor to neuroinflammation after intracerebral hemorrhage (ICH). Dectin-1 is …

Hematoma clearance, which is achieved largely by phagocytosis of erythrocytes in the
hemorrhagic brain, limits injury and facilitates recovery following intracerebral hemorrhage …

Background Pyroptosis is a programmed cell death mediated by inflammasomes. Previous
studies have reported that inhibition of neurokinin receptor 1 (NK1R) exerted …