The malaria-causing parasite, Plasmodium falciparum completely remodels its host red
blood cell (RBC) through the export of several hundred parasite proteins, including …

Virulence in pathogenic protozoa is often tied to secretory processes such as the expression
of adhesins on parasite surfaces or the secretion of proteases to assisted in tissue invasion …

Plasmodium falciparum extensively remodels human erythrocytes by exporting hundreds of
parasite proteins. This remodeling is closely linked to the Plasmodium virulence-related …

Plasmodium falciparum undergoes a series of asexual replications in human erythrocytes
after infection, which are effective targets for combatting malaria. Here, we report roles of an …

Plasmodium falciparum accounts for the majority of malaria deaths, due to pathology
provoked by the ability of infected erythrocytes to adhere to vascular endothelium within …

Plasmodium species cause malaria, and in the instance of Plasmodium falciparum is
responsible for a societal burden of over 600,000 deaths annually. The symptoms and …

Plasmodium falciparum undergoes a series of asexual replications in human erythrocytes
after infection, which are effective targets for combatting malaria. Here, we report roles of an …

PEXEL-positive protein Rifin was decreased by the expression of the inactive mutant of
PfRab1b or PfArf1. These data indicate that the transport of PfAK2 and Rifin were …

Newly synthesized secretory proteins are transported from the endoplasmic reticulum (ER)
via the Golgi apparatus to be delivered to plasma membrane. Sar1 GTPase plays a critical …

The deadly human malaria-causing parasite, Plasmodium falciparum relies on its capacity to
completely remodel its host red blood cell (RBC) through the export of hundreds of parasite …