Plasmodium parasites, the causative agents of malaria, have developed elaborate
strategies that they use to survive and thrive within different intracellular environments …

Exported proteins of the malaria parasite Plasmodium falciparum interact with proteins of the
erythrocyte membrane and induce substantial changes in the morphology, physiology and …

Cell-cell communication is an important mechanism for information exchange promoting cell
survival for the control of features such as population density and differentiation. We …

During the blood stages of malaria, several hundred parasite-encoded proteins are exported
beyond the double-membrane barrier that separates the parasite from the host cell …

A major part of virulence for Plasmodium falciparum malaria infection, the most lethal
parasitic disease of humans, results from increased rigidity and adhesiveness of infected …

Understanding the targets and mechanisms of human immunity to malaria caused by
Plasmodium falciparum is crucial for advancing effective vaccines and developing tools for …

Malaria blood stage parasites export a large number of proteins into their host erythrocyte to
change it from a container of predominantly hemoglobin optimized for the transport of …

The deadliest form of human malaria is caused by the protozoan parasite Plasmodium
falciparum affecting millions worldwide every year. P. falciparum virulence is attributed to its …

One of the most fascinating and remarkable features of Plasmodium parasites, which cause
malaria, is their choice of erythrocytes as the principal host cells in which to reside during …

Malaria parasites modify their host cell, the mature human erythrocyte. We are interested in
the molecules mediating these processes, and have recently described a family of parasite …