[HTML][HTML] Novel insights into the potential mechanisms of N6-methyladenosine RNA modification on sepsis-induced cardiovascular dysfunction: an update summary on …

Y Wang, M Xu, P Yue, D Zhang, J Tong… - Frontiers in cell and …, 2021 - frontiersin.org
Sepsis is a life-threatening organ dysfunction caused by a host's dysfunctional response to
infection. As is known to all, septic heart disease occurs because pathogens invading the …

[HTML][HTML] Detecting critical functional ingredients group and mechanism of Xuebijing Injection in Treating Sepsis

Q Wu, C Yin, Y Li, J Cai, H Yang, Y Huang… - Frontiers in …, 2021 - frontiersin.org
Sepsis is a systemic inflammatory reaction caused by various infectious or noninfectious
factors, which can lead to shock, multiple organ dysfunction syndrome, and death. It is one of …

Cardioprotective Potential of Celastrol in Sepsis-Induced Cardiotoxicity; Mouse Model of Endotoxemia

FA Ghafil, ES Hassan, ND Aziz, MM Salim… - Iranian Journal of War …, 2023 - ijwph.ir
Aims: The study aimed to assess the cardioprotective potential of celastrol against cardiac
injury induced by sepsis via amelioration of IL6, TNF, TLR4, IL10, F2-isoprostane, cardiac …

Role of activating the nuclear factor kappa B signaling pathway in the development of septic cardiomyopathy in rats with sepsis

XR Xing, LP Luo, YL Li, YW Guo… - … and Health Care, 2023 - content.iospress.com
BACKGROUND: Despite advances in the treatment of sepsis over time, this condition
remains both a serious threat and a cause of death among critical patients. OBJECTIVE …

The Role of Regulator of G Protein Signaling 2 in Inflammatory Cytokine Release in Endotoxemia in Mice

XT Ma - 2020 - search.proquest.com
In sepsis, lipopolysaccharide (LPS) activates toll-like receptor 4 to stimulate the release of
inflammatory cytokines (eg tumor necrosis factor-alpha, TNF-α), leading to cardiac …