Seizures are a well‐recognized and often prominent manifestation of autoimmune
encephalitic syndromes. Progress in detection of pathogenic neural autoantibodies has led …

Antibodies to glutamic acid decarboxylase (GAD) have been associated with several
neurological syndromes, including stiff-person syndrome, cerebellar ataxia and epilepsy …

The rapid expansion in the number of encephalitis disorders associated with autoantibodies
against neuronal proteins has led to an incremental increase in use of the term “autoimmune …

The field of autoimmune epilepsy has evolved substantially in the last few decades with
discovery of several neural autoantibodies and improved mechanistic understanding of …

Although seizures are frequent in all types of autoimmune encephalitis, the risk for chronic
epilepsy is dependent on the antigen: lower if located on the cell-surface, and higher if …

Epilepsy is seen historically as a disease of aberrant neuronal signaling manifesting as
seizures. With the discovery of numerous auto‐antibodies and the subsequent growth in …

Antibodies against glutamic acid decarboxylase (GAD), originally linked to stiff person
syndrome (SPS), now denote the “GAD antibody-spectrum disorders”(GAD-SD) that also …

Temporal lobe epilepsy (TLE) is one of the syndromes linked to antibodies against glutamic
acid decarboxylase (GAD). It has been questioned whether 'limbic encephalitis with GAD …

Glutamic acid decarboxylase (GAD) antibody-associated encephalitis causes both acute
seizures and chronic epilepsy with predominantly temporal lobe onset. This condition is …

Seizures and epilepsy can result from various aetiologies, yet the underlying cause of
several epileptic syndromes remains unclear. In that regard, autoimmune-mediated …