Tubulovascular nitric oxide crosstalk: buffering of angiotensin II–induced medullary vasoconstriction
JG Dickhout, T Mori, AW Cowley Jr - Circulation research, 2002 - Am Heart Assoc
Studies were designed to determine the source of NO responsible for buffering of the
angiotensin II (Ang II)–mediated decrease of blood flow in the renal medulla. Intracellular …
angiotensin II (Ang II)–mediated decrease of blood flow in the renal medulla. Intracellular …