In a literature survey, Chernoff et al.(2017) dismissed the hypothesis that chronic exposure
to β-N-methylamino-L-alanine (BMAA) may be a risk factor for progressive …

Neurodegenerative diseases are influenced by environmental factors such as exposure to
toxins including the cyanotoxin β-N-methylamino-L-alanine (BMAA) that can bioaccumulate …

Cetaceans are well-regarded as sentinels for toxin exposure. Emerging studies suggest that
cetaceans can also develop neuropathological changes associated with neurodegenerative …

β-methylamino-L-alanine (BMAA) is a non-protein amino acid that has been implicated as a
risk factor for motor neurone disease (MND). BMAA is produced by a wide range of …

The neurotoxic alkaloid β-N-methyl-amino-l-alanine (BMAA) and related isomers, including
N-(2-aminoethyl glycine)(AEG), β-amino-N-methyl alanine (BAMA), and 2, 4-diaminobutyric …

Exposure to cyanotoxins has been linked to neurodegenerative diseases, including
amyotrophic lateral sclerosis, Alzheimer's, and Parkinson's disease. While the cyanotoxin β …

Introduction Cyanobacterial blooms produce toxins that may become aerosolized,
increasing health risks through inhalation exposures. Health related effects on the lower …

Environmental exposure to the amino acid β-methylamino-L-alanine (BMAA) was linked to
the high incidence of neurodegenerative disease first reported on the island of Guam in the …

Exposure to toxins produced by cyanobacteria (ie, cyanotoxins) is an emerging health
concern due to their increasing prevalence and previous associations with …

There is evidence that the environmental toxin β-N-methylamino-L-alanine (L-BMAA) may
be involved in neurodegenerative diseases. However, a number of controversies exist …