Ca2+/calmodulin-dependent protein kinase II (CaMKII) and long-term potentiation (LTP)
were discovered within a decade of each other and have been inextricably intertwined ever …

▪ Abstract Highly enriched in brain tissue and present throughout the body, Ca2+/calmodulin-
dependent protein kinase II (CaMKII) is central to the coordination and execution of Ca2+ …

Ca2+/calmodulin (CaM)-dependent protein kinase (CaMKII) is a ubiquitous mediator of
Ca2+-linked signalling that phosphorylates a wide range of substrates to co-ordinate and …

The dodecameric protein kinase CaMKII is expressed throughout the body. The alpha
isoform is responsible for synaptic plasticity and participates in memory through its …

Calcium/calmodulin-dependent protein kinase II (CaMKII) has diverse roles in virtually all
cell types and it is regulated by a plethora of mechanisms. Local changes in Ca2+ …

Calcium/calmodulin-dependent protein kinase II (CaMKII) is thought to be a critical mediator
of neuronal plasticity that links transiently triggered Ca2+ signals to persistent changes in …

Postsynaptic densities (PSDs) contain proteins that regulate synaptic transmission. We
determined the positions of calcium/calmodulin-dependent protein kinase II (CaMKII) and …

Ischemic insults on neurons trigger excessive, pathological glutamate release that causes
Ca 2+ overload resulting in neuronal cell death (excitotoxicity). The Ca 2+/calmodulin (CaM) …

The activity of Ca2+/calmodulin-dependent protein kinase II (CaMKII) plays an integral role
in regulating synaptic development and plasticity. We designed a live-cell-imaging approach …

Ca 2+/calmodulin (CaM)-dependent protein kinase II (CaMKII) is a major mediator of
physiological glutamate signaling involved in higher brain functions. Here, we show CaMKII …