Abstract Latent Epstein–Barr virus (EBV) infection has a substantial role in causing many
human disorders. The persistence of these viral genomes in all malignant cells, yet with the …

The tumor necrosis factor receptor (TNFR) superfamily represents a growing family, with
over 20 members having been identified thus far in mammalian cells. These proteins share …

Almost exactly twenty years after the discovery of Epstein-Barr virus (EBV), the latent
membrane protein 1 (LMP1) entered the EBV stage, and soon thereafter, it was recognized …

BCMA (B cell maturation) is a nonglycosylated integral membrane type I protein that is
preferentially expressed in mature B lymphocytes. Previously, we reported in a human …

Viral and microbial constituents contain specific motifs or pathogen-associated molecular
patterns (PAMPs) that are recognized by cell surface-and endosome-associated Toll-like …

The oncogenic Epstein–Barr virus (EBV)-encoded latent membrane protein 1 (LMP1) has
structural features and functions reminiscent of a constitutively active TNF family receptor …

Epstein-Barr virus (EBV) latency III infection converts B lymphocytes into lymphoblastoid cell
lines (LCLs) by expressing EBV nuclear and membrane proteins, EBNAs, and latent …

Tumor necrosis factor–α (TNF‐α) and TNF receptors (TNFR) are members of the growing
TNF ligand and receptor families that are involved in immune regulation. The present report …

Epstein-Barr virus infection of B lymphocytes elicits diverse host responses via well-adapted
transcriptional control dynamics. Consequently, this host-pathogen interaction provides a …

Epstein–Barr virus (EBV) transforms B lymphocytes into lymphoblastoid cell lines usurping
the Notch and tumor necrosis factor receptor pathways to effect transcription including NF-κB …