Current understanding of the molecular mechanisms of circulating permeability factor in focal segmental glomerulosclerosis

G Salfi, F Casiraghi, G Remuzzi - Frontiers in immunology, 2023 - frontiersin.org
The pathogenetic mechanisms underlying the onset and the post-transplant recurrence of
primary focal segmental glomerulosclerosis (FSGS) are complex and remain yet to be fully …

The complement C3a and C3a receptor pathway in kidney diseases

S Gao, Z Cui, M Zhao - Frontiers in immunology, 2020 - frontiersin.org
The pathogenesis of some kidney diseases is closely associated with complement
activation, where the C3a/C3a receptor (C3aR) might play a crucial role. C3a/C3aR has …

[HTML][HTML] SGLT2 inhibitor dapagliflozin limits podocyte damage in proteinuric nondiabetic nephropathy

P Cassis, M Locatelli, D Cerullo, D Corna, S Buelli… - JCI insight, 2018 - ncbi.nlm.nih.gov
Abstract Sodium-glucose cotransporter 2 (SGLT2) inhibitors have pleiotropic properties
beyond blood glucose–lowering effects and modify important nonglycemic pathways …

SARS-CoV-2 spike protein induces lung endothelial cell dysfunction and thrombo-inflammation depending on the C3a/C3a receptor signalling

L Perico, M Morigi, A Pezzotta, M Locatelli, B Imberti… - Scientific Reports, 2023 - nature.com
The spike protein of Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2) can
interact with endothelial cells. However, no studies demonstrated the direct effect of the …

[HTML][HTML] C3a receptor blockade protects podocytes from injury in diabetic nephropathy

M Morigi, L Perico, D Corna, M Locatelli, P Cassis… - Jci Insight, 2020 - ncbi.nlm.nih.gov
Renal activation of the complement system has been described in patients with diabetic
nephropathy (DN), although its pathological relevance is still ill-defined. Here, we studied …

Loss of decay-accelerating factor triggers podocyte injury and glomerulosclerosis

A Angeletti, C Cantarelli, A Petrosyan… - Journal of Experimental …, 2020 - rupress.org
Kidney glomerulosclerosis commonly progresses to end-stage kidney failure, but
pathogenic mechanisms are still poorly understood. Here, we show that podocyte …

Therapeutic potential of stromal cells of non-renal or renal origin in experimental chronic kidney disease

C Rota, M Morigi, D Cerullo, M Introna… - Stem cell research & …, 2018 - Springer
Background Mesenchymal stromal cell (MSC)-based therapy is a promising strategy for
preventing the progression of chronic kidney disease (CKD), with the potential to induce …

Serum protein pattern associated with organ damage and lupus nephritis in systemic lupus erythematosus revealed by PEA immunoassay

A Petrackova, A Smrzova, P Gajdos, M Schubertova… - Clinical proteomics, 2017 - Springer
Background Systemic lupus erythematosus (SLE) is a remarkably heterogeneous
autoimmune disease. Despite tremendous efforts, our knowledge of serum protein patterns …

Shiga toxin triggers endothelial and podocyte injury: the role of complement activation

C Zoja, S Buelli, M Morigi - Pediatric nephrology, 2019 - Springer
Abstract Shiga toxin (Stx)-producing Escherichia coli (STEC) is the offending agent in post-
diarrhea-associated hemolytic uremic syndrome (HUS), a disorder characterized by …

Parietal epithelial cell dysfunction in crescentic glomerulonephritis

MN Wong, PL Tharaux, F Grahammer… - Cell and Tissue …, 2021 - Springer
Crescentic glomerulonephritis represents a group of kidney diseases characterized by rapid
loss of kidney function and the formation of glomerular crescents. While the role of the …