Food intake is a complex behavior that can occur or cease to occur for a multitude of
reasons. Decisions about where, when, what, and how much to eat are not merely reflexive …

The neural circuits controlling feeding and emotional behaviors are intricately and
reciprocally connected. Recent technological developments, including cell type-specific …

Feeding behavior is orchestrated by neural circuits primarily residing in the hypothalamus
and hindbrain. However, the relative influence of cognitive and emotional brain circuits to …

Stress can cause a variety of central nervous system disorders, which are critically mediated
by the γ‐aminobutyric acid (GABA) system in various brain structures. GABAergic neurons …

Hindbrain glucagon-like peptide 1 (GLP-1) neurons project to numerous forebrain areas,
including the lateral septum (LS). Using a fluorescently labeled GLP-1 receptor (GLP-1R) …

Binge eating disorder (BED), characterized by bingeing episodes and compulsivity, is the
most prevalent eating disorder; however, little is known about its neurobiological …

Abstract Glucagon-like peptide 1 (GLP-1) neurons of the caudal brainstem project to many
brain areas, including the lateral septum (LS), which has a known role in stress responses …

Phoenixin is a novel neuropeptide initially associated with reproductive functions, but
subsequently also with feeding behavior. Nesfatin-1 is also involved in the regulation of food …

Abstract The neuropeptide relaxin-3 (RLN3) binds with high affinity to its cognate receptor,
relaxin-family peptide receptor 3 (RXFP3), and with lower affinity to RXFP1, the cognate …

Arousal is a process that involves the activation of ascending neural pathways originating in
the rostral pons that project to the forebrain through the midbrain reticular formation to …