beta-cell function and viability in the spontaneously diabetic GK rat: information from the GK/Par colony.
B Portha, MH Giroix, P Serradas, MN Gangnerau… - Diabetes, 2001 - Am Diabetes Assoc
The GK rat model of type 2 diabetes is especially convenient to dissect the pathogenic
mechanism necessary for the emergence of overt diabetes because all adult rats obtained in …
mechanism necessary for the emergence of overt diabetes because all adult rats obtained in …
Effect of gliclazide treatment on insulin secretion and β-cell mass in non-insulin dependent diabetic Goto–Kakisaki rats
N Dachicourt, D Bailbé, MN Gangnerau… - European journal of …, 1998 - Elsevier
The Goto–Kakisaki rat is a genetic non-overweight model of non-insulin-dependent diabetes
mellitus. Adult Goto–Kakisaki rats exhibit a mild basal hyperglycaemia (11 mmol/l) with …
mellitus. Adult Goto–Kakisaki rats exhibit a mild basal hyperglycaemia (11 mmol/l) with …
[PDF][PDF] Defective glucose-dependent cytosolic Ca2+ handling in islets of GK and nSTZ rat models of type 2 diabetes
JC Marie, D Bailbe, E Gylfe, B Portha - Journal of endocrinology, 2001 - academia.edu
We examined to what extent the abnormal glucosedependent insulin secretion observed in
NIDDM (noninsulin-dependent diabetes mellitus) is related to alterations in the handling of …
NIDDM (noninsulin-dependent diabetes mellitus) is related to alterations in the handling of …
Restitution of defective glucose-stimulated insulin secretion in diabetic GK rat by acetylcholine uncovers paradoxical stimulatory effect of β-cell muscarinic receptor …
M Dolz, D Bailbé, MH Giroix, S Calderari… - Diabetes, 2005 - Am Diabetes Assoc
Because acetylcholine (ACh) is a recognized potentiator of glucose-stimulated insulin
release in the normal β-cell, we have studied ACh's effect on islets of the Goto-Kakizaki (GK) …
release in the normal β-cell, we have studied ACh's effect on islets of the Goto-Kakizaki (GK) …
Tissue inhibitor of metalloproteinase-1 mRNA production and protein secretion are induced by interleukin-1β in 3T3-L1 adipocytes
S Weise, S Kralisch, G Sommer… - Journal of …, 2008 - joe.bioscientifica.com
The mechanism by which ethane 1, 2-dimethanesulfonate (EDS) selectively kills Leydig
cells is poorly understood. To characterize further the cell-specific actions of EDS, we …
cells is poorly understood. To characterize further the cell-specific actions of EDS, we …
Cytosolic calcium handling in islets of normal Wistar and diabetic Goto Kakizaki rats in the presence of glucose and truncated glucagon-like peptide 1 (7-36) amide
JC Marie, D Bailbe - Annals of the New York Academy of …, 2000 - pubmed.ncbi.nlm.nih.gov
Cytosolic calcium handling in islets of normal Wistar and diabetic Goto Kakizaki rats in the
presence of glucose and truncated glucagon-like peptide 1 (7-36) amide Cytosolic calcium …
presence of glucose and truncated glucagon-like peptide 1 (7-36) amide Cytosolic calcium …
Stimulus-secretion coupling in pancreatic β-cells of healthy and diabetic rats in tissue slice preparation
T Rose - 2006 - ediss.uni-goettingen.de
Eines der ersten Ereignisse in der Entwicklung von Diabetes des Typs 2 ist eine verringerte
Kapazität der β-Zellen des Pankreas, Insulin als Antwort auf eine Stimulation mit Glukose zu …
Kapazität der β-Zellen des Pankreas, Insulin als Antwort auf eine Stimulation mit Glukose zu …
[引用][C] Stimulus-secretion coupling in pancreatic β-cells [beta-cells] of healthy and diabetic rats in tissue slice preparation
T Rose - 2006 - Göttingen, Univ., Diss., 2006
[引用][C] JEAN-CLAUDE MARIE, AND DANIELLE BAILBE
N Wistar, DG Kakizaki - VIP, PACAP …, 2000 - New York Academy of Sciences