There has long been a great concern with growing anthropogenic contaminants and their
ecological and toxicological effects on living organisms and the surrounding environment for …

Abstract Polycyclic Aromatic Hydrocarbons (PAHs) represent persistent environmental
pollutants ubiquitously distributed in the environment. Their presence alongside various …

Decreased lung function is an early hazard of respiratory damage from fine particulate
matter (PM 2.5) exposure. Limited studies have explored the association between PM 2.5 …

Central nervous system (CNS) trauma, including traumatic brain injury and spinal cord
injury, has a high rate of disability and mortality, and effective treatment is currently lacking …

Background: Humans are exposed to mixtures of toxicants that can impact several biological
pathways. We investigated the associations between multiple classes of toxicants and an …

Dysregulation of gap junction intercellular communication (GJIC) is recognized as one of the
key hallmarks for identifying non-genotoxic carcinogens (NGTxC). Currently, there is a …

Gap junctional intercellular communication (GJIC) is a vital cellular process required for
maintenance of tissue homeostasis. In vitro assessment of GJIC represents valuable …

The WHO classified air pollution as a human lung carcinogen and polycyclic aromatic
hydrocarbons (PAHs) are components of both indoor (eg, tobacco smoke and cookstoves) …

The lung inflammatory damage could result from the nickel oxide nanoparticles (NiO NPs),
in which the underlying mechanism is still unclear. This article explored the roles of long …

Polycyclic aromatic hydrocarbon (PAH) exposure is a cause of chronic inflammation. The
effect of PAHs on bioactive lipid mediators involved in the inflammatory process remains …