TWEAK is a multifunctional cytokine that controls many cellular activities including
proliferation, migration, differentiation, apoptosis, angiogenesis and inflammation. TWEAK …

Our immune system performs the vital function of recognizing and eliminating invading
pathogens and malignancies. There is an increasing appreciation that the immune system …

First described as a weak apoptosis inducer, the TNF superfamily ligand TWEAK has since
emerged as a cytokine that regulates multiple cellular responses, including proinflammatory …

Tumor necrosis factor (TNF)-like weak inducer of apoptosis (TWEAK) is a member of the
TNF superfamily that has been shown to induce angiogenesis, apoptosis in tumor cells, and …

Background & Aims Cholangiocarcinoma (CCA) is a cancer of the hepatic bile ducts that is
rarely resectable and is associated with poor prognosis. Tumour necrosis factor-like weak …

TWEAK (tumor necrosis factor–related weak inducer of apoptosis), a member of the tumor
necrosis factor superfamily, acts on cells by binding to its only receptor named Fn14 …

TWEAK (TNF-like weak inducer of apoptosis) is a TNF superfamily member implicated in
several mechanisms. Although fibroblast growth factor inducible 14 (Fn14)/TweakR has …

Introduction TNF-like weak inducer of apoptosis (TWEAK) has been implicated as a
mediator of chronic inflammatory processes via prolonged activation of the NF-κB pathway …

For successful nerve regeneration, a coordinated shift in gene expression pattern must
occur in axotomized neurons. To identify genes participating in axonal regeneration, we …

TNF-like weak inducer of apoptosis (TWEAK) is a member of the TNF superfamily of
cytokines. In addition to binding and activating the fibroblast growth factor–inducible 14 …