Tumour necrosis factor gene polymorphism and disease prevalence
Tumour necrosis factor (TNF), an important proinflammatory cytokine, plays a role in the
regulation of cell differentiation, proliferation and death, as well as in inflammation, innate …
regulation of cell differentiation, proliferation and death, as well as in inflammation, innate …
An understanding of the genetic basis of asthma
M Bijanzadeh, PA Mahesh… - Indian Journal of …, 2011 - journals.lww.com
Asthma is the most common chronic childhood disease in developed nations and its
prevalence has increased in the world over the last 25 years. It is a complex disease with …
prevalence has increased in the world over the last 25 years. It is a complex disease with …
Quantitative and causal analysis for inflammatory genes and the risk of Parkinson's disease
M Yi, J Li, S Jian, B Li, Z Huang, L Shu… - Frontiers in …, 2023 - frontiersin.org
Background The dysfunction of immune system and inflammation contribute to the
Parkinson's disease (PD) pathogenesis. Cytokines, oxidative stress, neurotoxin and …
Parkinson's disease (PD) pathogenesis. Cytokines, oxidative stress, neurotoxin and …
Polymorphism of tumor necrosis factor alpha (TNF-alpha) gene promoter, circulating TNF-alpha level, and cardiovascular risk factor for ischemic stroke
G Cui, H Wang, R Li, L Zhang, Z Li, Y Wang… - Journal of …, 2012 - Springer
Background Tumor necrosis factor-α (TNF-α) is one of the most typical pro-inflammatory
cytokines with both beneficial and destructive properties for the central nervous system …
cytokines with both beneficial and destructive properties for the central nervous system …
Leprosy as a genetic disease
Leprosy (Hansen's disease) is a human infectious disease whose etiological agent,
Mycobacterium leprae, was identified by GHA Hansen in the 19th century. Despite the high …
Mycobacterium leprae, was identified by GHA Hansen in the 19th century. Despite the high …
Genetic, immune-inflammatory, and oxidative stress biomarkers as predictors for disability and disease progression in multiple sclerosis
The aim of this study was to evaluate the TNFβ Nco I polymorphism (rs909253) and immune-
inflammatory, oxidative, and nitrosative stress (IO&NS) biomarkers as predictors of disease …
inflammatory, oxidative, and nitrosative stress (IO&NS) biomarkers as predictors of disease …
Tumor necrosis factor–α is a common genetic risk factor for asthma, juvenile rheumatoid arthritis, and systemic lupus erythematosus in a Mexican pediatric population
S JimÊnez-Morales, R VelÂzquez-Cruz… - Human immunology, 2009 - Elsevier
There is a great deal of evidence that points to the association of the tumor necrosis factor-
alpha (TNF-α) gene as a common genetic factor in the pathogenesis of diseases that are …
alpha (TNF-α) gene as a common genetic factor in the pathogenesis of diseases that are …
Association between genetic variants of IL-1β, IL-6 and TNF-α cytokines and cognitive performance in the elderly general population of the MEMO-study
This study is to investigate the associations between specific polymorphisms in three
cytokine genes and domains of cognitive functioning in a population based study in the …
cytokine genes and domains of cognitive functioning in a population based study in the …
Lipopolysaccharide (LPS) exposure differently affects allergic asthma exacerbations and its amelioration by intranasal curcumin in mice
Aim Lipopolysaccharide (LPS) is ubiquitous in the environment and can therefore,
exacerbate allergic responses. Studies have suggested immunoregulatory effects of LPS …
exacerbate allergic responses. Studies have suggested immunoregulatory effects of LPS …
Association of TNF‐α with severe respiratory syncytial virus infection and bronchial asthma
B Puthothu, S Bierbaum, MV Kopp… - Pediatric allergy and …, 2009 - Wiley Online Library
Tumor necrosis factor (TNF‐) α is a proinflammatory cytokine that is important in the innate
host defence and thus in the defence of infectious agents. However, in excess it provokes …
host defence and thus in the defence of infectious agents. However, in excess it provokes …