Metastasis is the main cause of cancer-related mortality. Although the actual process of
metastasis remains largely elusive, epithelial-mesenchymal transition (EMT) has been …

Epithelial–mesenchymal transition (EMT) is a reversible cellular process, characterized by
changes in gene expression and activation of proteins, favoring the trans-differentiation of …

Recent investigations have emphasized the role of aberrant expression of microRNAs
(miRNAs) in progression of almost all types of cancers. Exosomes, membrane-enclosed …

Vasculogenic mimicry (VM) is the formation of vascular channels lacking endothelial cells.
These channels are lined by tumor cells with cancer stem cell features, positive for periodic …

The epithelial-to-mesenchymal transition (EMT) is a vital control point in metastatic breast
cancer (MBC). TWIST1, SNAIL1, SLUG, and ZEB1, as key EMT-inducing transcription factors …

The prognostic value of E-cadherin expression in patients with breast cancer has been
studied for years, yet results remain controversial. We thus performed a comprehensive …

More than 90% of cancer‐related deaths are caused by metastasis. Epithelial‐to‐
Mesenchymal Transition (EMT) causes tumor cell dissemination while the reverse process …

Twist, a highly conserved basic Helix-Loop-Helix transcription factor, functions as a major
regulator of epithelial-mesenchymal transition (EMT) and tumor metastasis. In different cell …

The oncogenic MUC1-C protein and the TWIST1 epithelial–mesenchymal transition
transcription factor (EMT-TF) are aberrantly expressed in triple-negative breast cancer …

Background Epithelial-mesenchymal transition (EMT) is a dedifferentiation process that
mainly involves in mesenchymal marker upregulation, epithelial maker downregulation and …