Human papillomaviruses (HPVs) cause diseases ranging from benign warts to invasive
cancers. HPVs infect epithelial cells and their replication cycle is tightly linked with the …

Human papillomaviruses (HPV) are the causative agents of cervical cancer and are also
associated with other genital malignancies, as well as an increasing number of head and …

Human papillomavirus infection is associated with the development of malignant and benign
neoplasms. Approximately 40 viral types can infect the anogenital mucosa and are …

Regulation of human papillomavirus (HPV) gene expression is tightly linked to differentiation
of the keratinocytes the virus infects. HPV late gene expression is confined to the cells in the …

The life cycle of human papillomavirus (HPV) depends on keratinocyte differentiation as the
virus modulates and takes advantage of cellular pathways to replicate its genome and …

The ubiquitous host protein, CCCTC-binding factor (CTCF), is an essential regulator of
cellular transcription and functions to maintain epigenetic boundaries, stabilise chromatin …

Human Papillomaviruses (HPV) reproduce in stratified epithelia by establishing a reservoir
of low-level infection in the dividing basal cells and restricting the production of viral particles …

Persistent high-risk human papillomavirus (HPV) infection is the major causal factor in
cervical and other anogenital cancers. Because there are currently no therapeutics capable …

Although many high-risk mucosal and cutaneous human papillomaviruses (HPVs)
theoretically have the potential to synthesize L1 isoforms differing in length, previous …

The papillomavirus (PV) E2 protein is a critical regulator of viral transcription and genome
replication. We previously reported that tyrosine (Y) 138 of HPV-31 E2 is phosphorylated by …