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[HTML][HTML] BET inhibition induces vulnerability to MCL1 targeting through upregulation of fatty acid synthesis pathway in breast cancer

G Yan, A Luna, H Wang, B Bozorgui, X Li, M Sanchez… - Cell reports, 2022 - cell.com
Therapeutic options for treatment of basal-like breast cancers remain limited. Here, we
demonstrate that bromodomain and extra-terminal (BET) inhibition induces an adaptive …
被引用次数：3 相关文章

BET Inhibition Induces Vulnerability to MCL1 Targeting Through Upregulation of Fatty Acid Synthesis Pathway in Breast Cancer

G Yan, A Luna, H Wang, B Bozorgui, X Li, M Sanchez… - papers.ssrn.com
The development of effective targeted therapies for the treatment of basal-like breast
cancers remains challenging. Here, we demonstrate that cellular stress engendered by BET …

BET inhibition induces vulnerability to MCL1 targeting through upregulation of fatty acid synthesis pathway in breast cancer

G Yan, A Luna, H Wang, B Bozorgui, X Li… - Cell …, 2022 - pubmed.ncbi.nlm.nih.gov
Therapeutic options for treatment of basal-like breast cancers remain limited. Here, we
demonstrate that bromodomain and extra-terminal (BET) inhibition induces an adaptive …

BET inhibition induces vulnerability to MCL1 targeting through upregulation of fatty acid synthesis pathway in breast cancer

G Yan, A Luna, H Wang, B Bozorgui, X Li… - Cell …, 2022 - ohsu.elsevierpure.com
Therapeutic options for treatment of basal-like breast cancers remain limited. Here, we
demonstrate that bromodomain and extra-terminal (BET) inhibition induces an adaptive …

BET inhibition induces vulnerability to MCL1 targeting through upregulation of fatty acid synthesis pathway in breast cancer.

G Yan, A Luna, H Wang, B Bozorgui, X Li, M Sanchez… - Cell Reports, 2022 - europepmc.org
Therapeutic options for treatment of basal-like breast cancers remain limited. Here, we
demonstrate that bromodomain and extra-terminal (BET) inhibition induces an adaptive …
[HTML] sciencedirect.com

[HTML][HTML] BET inhibition induces vulnerability to MCL1 targeting through upregulation of fatty acid synthesis pathway in breast cancer

G Yan, A Luna, H Wang, B Bozorgui, X Li, M Sanchez… - Cell Reports, 2022 - Elsevier
Therapeutic options for treatment of basal-like breast cancers remain limited. Here, we
demonstrate that bromodomain and extra-terminal (BET) inhibition induces an adaptive …

BET inhibition induces vulnerability to MCL1 targeting through upregulation of fatty acid synthesis pathway in breast cancer

G Yan, A Luna, H Wang, B Bozorgui… - Cell …, 2022 - mdanderson.elsevierpure.com
Therapeutic options for treatment of basal-like breast cancers remain limited. Here, we
demonstrate that bromodomain and extra-terminal (BET) inhibition induces an adaptive …
[HTML] nih.gov

[HTML][HTML] BET inhibition induces vulnerability to MCL1 targeting through upregulation of fatty acid synthesis pathway in breast cancer

G Yan, A Luna, H Wang, B Bozorgui, X Li, M Sanchez… - Cell reports, 2022 - ncbi.nlm.nih.gov
Therapeutic options for treatment of basal-like breast cancers remain limited. Here, we
demonstrate that bromodomain and extra-terminal (BET) inhibition induces an adaptive …

BET inhibition induces vulnerability to MCL1 targeting through upregulation of fatty acid synthesis pathway in breast cancer.

G Yan, A Luna, H Wang, B Bozorgui, X Li, M Sanchez… - Cell Reports, 2022 - europepmc.org
Therapeutic options for treatment of basal-like breast cancers remain limited. Here, we
demonstrate that bromodomain and extra-terminal (BET) inhibition induces an adaptive …