Hnf1α (MODY3) controls tissue-specific transcriptional programs and exerts opposed effects on cell growth in pancreatic islets and liver

JM Servitja, M Pignatelli, MA Maestro… - … and cellular biology, 2009 - Taylor & Francis
Heterozygous HNF1A mutations cause pancreatic-islet β-cell dysfunction and monogenic
diabetes (MODY3). Hnf1α is known to regulate numerous hepatic genes, yet knowledge of …
被引用次数：154 相关文章

Hnf1α (MODY3) Controls Tissue-Specific Transcriptional Programs and Exerts Opposed Effects on Cell Growth in Pancreatic Islets and Liver

JM Servitja, M Pignatelli, MA Maestro… - … and Cellular Biology, 2009 - Am Soc Microbiol
Heterozygous HNF1A mutations cause pancreatic-islet β-cell dysfunction and monogenic
diabetes (MODY3). Hnf1α is known to regulate numerous hepatic genes, yet knowledge of …

Hnf1α (MODY3) Controls Tissue-Specific Transcriptional Programs and Exerts Opposed Effects on Cell Growth in Pancreatic Islets and Liver

JM Servitja, M Pignatelli, MA Maestro… - Molecular and Cellular …, 2009 - cir.nii.ac.jp
抄録< jats: title> ABSTRACT</jats: title>< jats: p> Heterozygous< jats: italic> HNF1A</jats:
italic> mutations cause pancreatic-islet β-cell dysfunction and monogenic diabetes …

Hnf1alpha (MODY3) controls tissue-specific transcriptional programs and exerts opposed effects on cell growth in pancreatic islets and liver

JM Servitja, M Pignatelli, MA Maestro… - Molecular and …, 2009 - pubmed.ncbi.nlm.nih.gov
Heterozygous HNF1A mutations cause pancreatic-islet beta-cell dysfunction and monogenic
diabetes (MODY3). Hnf1alpha is known to regulate numerous hepatic genes, yet knowledge …

Hnf1alpha (MODY3) controls tissue-specific transcriptional programs and exerts opposed effects on cell growth in pancreatic islets and liver.

J Servitja, M Pignatelli, M Maestro… - Molecular and cellular …, 2009 - ora.ox.ac.uk
Heterozygous HNF1A mutations cause pancreatic-islet beta-cell dysfunction and monogenic
diabetes (MODY3). Hnf1alpha is known to regulate numerous hepatic genes, yet knowledge …
[HTML] nih.gov

[HTML][HTML] Hnf1α (MODY3) Controls Tissue-Specific Transcriptional Programs and Exerts Opposed Effects on Cell Growth in Pancreatic Islets and Liver

JM Servitja, M Pignatelli, MÁ Maestro… - … and Cellular Biology, 2009 - ncbi.nlm.nih.gov
Heterozygous HNF1A mutations cause pancreatic-islet β-cell dysfunction and monogenic
diabetes (MODY3). Hnf1α is known to regulate numerous hepatic genes, yet knowledge of …

Hnf1alpha (MODY3) controls tissue-specific transcriptional programs and exerts opposed effects on cell growth in pancreatic islets and liver.

JM Servitja, M Pignatelli, MA Maestro… - … and Cellular Biology, 2009 - europepmc.org
Heterozygous HNF1A mutations cause pancreatic-islet β-cell dysfunction and monogenic
diabetes (MODY3). Hnf1α is known to regulate numerous hepatic genes, yet knowledge of …
[PDF] researchgate.net

[PDF][PDF] Hnf1α (MODY3) Controls Tissue-Specific Transcriptional Programs and Exerts Opposed Effects on Cell Growth in Pancreatic Islets and Liver

JM Servitja, M Pignatelli, MA Maestro… - MOLECULAR AND …, 2009 - researchgate.net
Heterozygous HNF1A mutations cause pancreatic-islet-cell dysfunction and monogenic
diabetes (MODY3). Hnf1 is known to regulate numerous hepatic genes, yet knowledge of its …
[PDF] academia.edu

[PDF][PDF] Hnf1α (MODY3) Controls Tissue-Specific Transcriptional Programs and Exerts Opposed Effects on Cell Growth in Pancreatic Islets and Liver

JM Servitja, M Pignatelli, MA Maestro… - MOLECULAR AND …, 2009 - academia.edu
Heterozygous HNF1A mutations cause pancreatic-islet-cell dysfunction and monogenic
diabetes (MODY3). Hnf1 is known to regulate numerous hepatic genes, yet knowledge of its …

Hnf1alpha (MODY3) controls tissue-specific transcriptional programs and exerts opposed effects on cell growth in pancreatic islets and liver.

JM Servitja, M Pignatelli, MA Maestro… - … and Cellular Biology, 2009 - europepmc.org
Heterozygous HNF1A mutations cause pancreatic-islet β-cell dysfunction and monogenic
diabetes (MODY3). Hnf1α is known to regulate numerous hepatic genes, yet knowledge of …