[HTML][HTML] Mechanistic rationale for inhibition of poly (ADP-ribose) polymerase in ETS gene fusion-positive prostate cancer

JC Brenner, B Ateeq, Y Li, AK Yocum, Q Cao… - Cancer cell, 2011 - cell.com
Recurrent fusions of ETS genes are considered driving mutations in a diverse array of
cancers, including Ewing's sarcoma, acute myeloid leukemia, and prostate cancer. We …

Mechanistic rationale for inhibition of poly (ADP-ribose) polymerase in ETS gene fusion-positive prostate cancer.

JC Brenner, B Ateeq, Y Li, AK Yocum, Q Cao… - Cancer Cell, 2011 - europepmc.org
Recurrent fusions of ETS genes are considered driving mutations in a diverse array of
cancers, including Ewing's sarcoma, acute myeloid leukemia, and prostate cancer. We …

Mechanistic rationale for inhibition of Poly (ADP-Ribose) Polymerase in ETS gene fusion positive prostate cancer

JC Brenner, B Ateeq, Y Li, A Yocum, Q Cao, I Asangani… - Cancer Research, 2011 - AACR
Recurrent fusions of ETS genes are considered driving mutations in a diverse array of
cancers including Ewing's sarcoma, acute myeloid leukemia, and epithelial tumors such as …

Mechanistic rationale for inhibition of poly (ADP-ribose) polymerase in ETS gene fusion-positive prostate cancer

JC Brenner, B Ateeq, Y Li, AK Yocum, Q Cao… - Cancer …, 2011 - pubmed.ncbi.nlm.nih.gov
Recurrent fusions of ETS genes are considered driving mutations in a diverse array of
cancers, including Ewing's sarcoma, acute myeloid leukemia, and prostate cancer. We …

Mechanistic Rationale for Inhibition of Poly (ADP-Ribose) Polymerase in ETS Gene Fusion-Positive Prostate Cancer

JC Brenner, B Ateeq, Y Li, AK Yocum, Q Cao… - Cancer Cell, 2011 - academia.edu
(Cancer Cell 19, 664–678; May 17, 2011) In Figure 3B of this article, the VCAP and the
control siRNA images are identical. This was the result of a mistake in constructing the …

[引用][C] Mechanistic Rationale for Inhibition of Poly (ADP-Ribose) Polymerase in ETS Gene Fusion-Positive Prostate Cancer

JC Brenner, B Ateeq, Y Li, AK Yocum, Q Cao… - Cancer Cell, 2011 - cir.nii.ac.jp
Mechanistic Rationale for Inhibition of Poly(ADP-Ribose) Polymerase in ETS Gene Fusion-Positive
Prostate Cancer | CiNii Research CiNii 国立情報学研究所 学術情報ナビゲ恁縖サイニィ] 詳細へ …

Mechanistic Rationale for Inhibition of Poly (ADP-Ribose) Polymerase in ETS Gene Fusion-Positive Prostate Cancer

JC Brenner, B Ateeq, Y Li, AK Yocum, Q Cao… - Cancer Cell, 2011 - infona.pl
Recurrent fusions of ETS genes are considered driving mutations in a diverse array of
cancers, including Ewing's sarcoma, acute myeloid leukemia, and prostate cancer. We …

Mechanistic Rationale for Inhibition of Poly (ADP-Ribose) Polymerase in ETS Gene Fusion-Positive Prostate Cancer

JC Brenner, B Ateeq, Y Li, AK Yocum, Q Cao… - Cancer Cell, 2011 - repository.ias.ac.in
Recurrent fusions of ETS genes are considered driving mutations in a diverse array of
cancers, including Ewing's sarcoma, acute myeloid leukemia, and prostate cancer. We …

[HTML][HTML] Mechanistic Rationale for Inhibition of Poly (ADP-Ribose) Polymerase in ETS Gene Fusion-Positive Prostate Cancer

JC Brenner, B Ateeq, Y Li, AK Yocum, Q Cao… - Cancer Cell, 2011 - Elsevier
Recurrent fusions of ETS genes are considered driving mutations in a diverse array of
cancers, including Ewing's sarcoma, acute myeloid leukemia, and prostate cancer. We …

Mechanistic Rationale for Inhibition of Poly (ADP-Ribose) Polymerase in ETS Gene Fusion-Positive Prostate Cancer

JC Brenner, B Ateeq, Y Li, AK Yocum, Q Cao… - Cancer …, 2011 - pure.johnshopkins.edu
Recurrent fusions of ETS genes are considered driving mutations in a diverse array of
cancers, including Ewing's sarcoma, acute myeloid leukemia, and prostate cancer. We …