Background Smoking and stress, pancreatic cancer (PanCa) risk factors, stimulate
nitrosamine 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) and catecholamines …

The stimulation of β2-adrenoceptor, which is a major mediator for chronic stress-induced
cancers, has been implicated in the progression in the number of cancer cells, including …

Objectives: Propranolol inhibited pancreatic cancer cell proliferation by blocking signaling
through the β-adrenoceptor. We hypothesized that propranolol may suppress pancreatic …

Smoking and chronic stress are well-documented risk factors that are associated with β-
adrenoceptors in the development of pancreatic cancer. Stimulation of β-adrenoceptors can …

Results PANC-1 cells expressed both β1-AR and β2-AR protein. Exposure of PANC-1 cells
to increasing concentrations of NE stimulated cell proliferation in a dose-dependent fashion …

Results Non-selective β-ARs agonist Isoproterenol (ISO) significantly increased cell
proliferation via β-ARs in a dose-dependent manner, with concomitant activation of …

AIM: To discuss the expression of α-adrenoreceptors in pancreatic cancer cell lines PC-2
and PC-3 and the effects of α 1-and α 2-adrenoreceptor antagonists, yohimbine and urapidil …

In a recent clinical observation, the growth of endothelial tumors, such as nasopharyngeal
carcinoma, was repressed by the non-selective-adrenergic antagonist propranolol. In this …

Pancreatic cancer is the fourth leading cause of cancer deaths in developed countries.
Smoking is an established risk factor for this malignancy but the underlying mechanisms are …

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