… Currently we do not know whether estrogen increases the risk of arterial thrombotic events.
… that estrogen may cause increased apoptosis in human coronary artery endothelial cells (…

… by which estrogen promotes apoptosis in endothelial cells and … /Fas ligand (FasL) in human
coronary artery endothelial cells (… increase is associated with an increase in Fas expression. …

… of coronary artery disease. A … apoptosis in cells bearing its receptor, Fas, primarily cells of
the immune system. It was recently shown that FasL is expressed by arterial endothelial cells (…

… significantly after menopause, with loss of cardiovascular protection attributed to estrogen
… endothelial cells by up-regulating Fas and Fas ligand expression. J Clin Endocrinol Metab …

… [43]] have reported that 17β-estradiol increased Fas-L and Fas expression in cultured
human coronary artery endothelial cells, thereby increasing their apoptosis. Tibolone is a 19-…

… First, we showed that hypoxia stimulated sFasL release by up-regulating FasL expression
in EC. Second and most importantly, sFasL released from EC inhibited hypoxia-induced EC …

… regulatory pathways are also reported to down-regulate the early processes of neutrophil-binding
activity that precede apoptosis in human retinal pigment epithelial cells infected by …

… Endothelial growth and apoptosis are involved in endothelial repair and function. Disruption
… We previously demonstrated that the GLP-1 receptor is expressed in HCAECs and that GLP-…

… This latter hypothesis would be supported by our findings that with downregulation of ERα
receptor expression, estrogen's effects on ERK and apoptosis become stimulatory, reflecting …

… that cultured human coronary artery endothelial cells treated with … an increase in Fas and
soluble FasL (sFasL) expression (25) … endothelial cells (HCAEC) by up-regulating Fas and Fas …