The response of the regional cerebral blood flow (rCBF) to brain topical superfusion of 20 m
M K+ was characterized in a closed cranial window preparation in barbiturate anesthetized …

We tested the hypothesis that the CBF response to extracellular acidosis is mediated by
nitric oxide (NO). A closed cranial window, superfused with artificial CSF (aCSF), was …

We investigated the combined effect of increased brain topical K+ concentration and
reduction of the nitric oxide (NO.) level caused by nitric oxide scavenging or nitric oxide …

Background and Purpose—Animal studies suggest that nitric oxide (NO) is important in
basal cerebral blood flow (CBF) regulation and that it may mediate the vasodilatory …

Background—From animal studies it emerged that nitric oxide is important for the
modulation of CO2-mediated cerebral blood flow (CBF chemoregulation) but not for the …

The aim of the present study was to determine whether neuronal nitric oxide synthase
(nNOS)-derived nitric oxide (NO) plays a permissive role in the regulation of cerebral blood …

Hypoxemia and anemia are associated with increased CBF, but the mechanisms that link
the changes in Pao2 or arterial O2 content (Cao2) with CBF are unclear. These experiments …

The importance of nitric oxide (NO) for CBF variations associated with arterial carbon
dioxide changes was investigated in halothane-anesthetized rats by using an inhibitor of …

We investigated, using a direct, intravital microscopic technique, whether nitric oxide (NO)
from neuronal nitric oxide synthase (nNOS) plays a role in the cerebral capillary flow …

The role of nitric oxide (NO) synthesis in the cerebral hyperemic responses to hypercapnia
and hypoxia was investigated in anesthetized rats. Regional CBF (rCBF) measurements …