BACKGROUND Interleukin 1β (IL-1β) and its natural antagonist have been implicated in the
pathogenesis of inflammatory bowel disease (IBD). Both cytokines influence bone formation …

Bone loss in inflammatory bowel disease (IBD) is presumed to be mediated by inflammation.
Increased levels of the multifunctional cytokine IL-6 in inflammatory diseases have been …

Background & Aims: Although bone loss and osteoporosis are well-known long-term
sequelae of inflammatory bowel disease (IBD), the risk factors for increased bone loss have …

Background: Osteoporosis is an important cause of morbidity in patients with Crohn's
disease. The pathogenesis of reduced bone mineral density (BMD) is multifactorial. A range …

Celiac disease (CD) is associated with decreased bone mineral mass. Its pathogenesis is
multifactorial since both systemic and local mechanisms may play a role. Our objective was …

Abstract Background and Aim Patients with inflammatory bowel disease (IBD) are at a high
risk of low bone mineral density (BMD). Reportedly, clinical and genetic factors cause low …

Abstract Background Interleukin (IL)‐1β stimulates the proliferation and differentiation of
osteoclast precursors into mature osteoclasts. IL‐1B polymorphisms may influence the gene …

Background: Inflammatory bowel disease (IBD) patients are reported to have lower bone
density compared to healthy controls. There is limited consensus regarding factors affecting …

Objective: Osteoporosis is a common disorder with a strong genetic component. Our aim
was to investigate the correlations of the interleukin-1β (IL-1β) and interleukin-1 receptor …

Background Osteoporosis is a recognized complication of inflammatory bowel disease
(IBD). Aim To investigate the role of environmental factors and vitamin D receptor (VDR) …