Angiostensin II (Ang II) regulates the migration and proliferation of vascular smooth muscle
cells. Recent studies indicate that intermediate-conductance Ca2+-activated K+ (IKca) …

We investigated the effects of the vasoconstrictor angiotensin (Ang) II on the whole cell
inward rectifier K+ (Kir) current enzymatically isolated from small-diameter (< 100μm) …

The vasoconstrictor angiotensin II (ANG II) inhibits several types of K+ channels. We
examined the inhibitory mechanism of ANG II on voltage-gated K+ (KV) currents (I KV) …

The proliferation of cardiac fibroblasts is implicated in the pathogenesis of myocardial
remodeling and fibrosis. Intermediate-conductance calcium-activated K+ channels (KCa3. 1 …

We previously reported that the Ca2+-activated K+ channel (Kca-channel) in cultured
smooth muscle cells from porcine coronary artery was inhibited by protein kinase C (C …

ATP-sensitive K+ channels with small conductance (30 pS in symmetrical K+-rich solutions)
in porcine coronary artery smooth muscle cells were highly active at physiological …

Although the presence of intracellular angiotensin II (Ang II) and of Ang II-binding sites has
been reported, their roles in cell function have not been fully clarified. The purpose of the …

The effects of the vasoconstrictor angiotensin II (Ang II) on whole-cell ATP-sensitive K+
currents (IK, ATP) of smooth muscle cells isolated enzymatically from rat mesenteric arteries …

Angiotensin II (Ang II) is one of the most potent vasoconstrictor substances, yet
paradoxically, Ang II may dilate certain vascular beds via an undefined mechanism. Ang II …

Angiotensin II inhibits rat arterial KATP channels by inhibiting steady-state protein kinase A
activity and activating protein kinase Cε - PMC Back to Top Skip to main content NIH NLM Logo …